研究人员发现，在实验室关闭RAD51分子，杀死恶性胶质瘤细胞，提高了放射治疗的效果。
Researchers discovered that switching off the RAD51 molecule increased the effectiveness of radiotherapy to kill glioblastoma cells in the lab.
研究人员已经建议一些特殊种类的恶性胶质瘤细胞可以繁殖，复制出相同的拷贝从而更加的耐治疗。
Researchers had suggested that some specialised groups of glioblastoma cells can reproduce to make identical copies that are more resistant to treatment.
埃伯哈特和他的同事们研究了恶性胶质瘤的细胞系以发现γ-内分泌酶抑制剂的影响，这种药物可阻断促使肿瘤增长的Notch受体途径。
Eberhart and colleagues studied glioblastoma cell lines to investigate the effects of a gamma-secretase inhibitor, a medication that blocks the Notch receptor, on tumor growth.
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