模型组大鼠心肌细胞结构出现明显的病理改变。
The obvious pathological changes of cardiomyocyte structure were found in model group.
结论：心肌肽素通过抑制心肌细胞凋亡，减轻心肌细胞结构损伤发挥对大鼠心肌缺血再灌注损伤的保护作用。
CONCLUSION: Cardiomyopeptidin plays a role in protecting myocardial ischemia-reperfusion injury in rats by inhibiting myocardial apoptosis and alleviating myocardial structural lesions.
它们对于心肌细胞起结构支持作用，在生长和病理条件下，负责细胞外基质的合成。
They provide structural support for cardiac myocytes and are responsible for extracellular matrix synthesis in the heart during growth and pathophysiological conditions.
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