他希望制药公司现在能够设计基于这些基因的测试来找出那些疤痕生成或快或慢的患者，并开发进促进愈合的药物。
He hopes that a pharmaceutical firm will now devise tests based on these genes to identify those in whom scarring progresses rapidly or slowly, as well as drugs that improve healing.
他们发现MMP-2诱导疤痕屏障移除，并使得完整视网膜的健康供体细胞与小鼠受损视网膜建立新的联系。
They found that MMP-2 induced removal of the scar barrier and allowed healthy donor cells (of the whole retina) to make new connections with the damaged retinas in the mice.
一旦疤痕发生时，来自脂肪细胞不能吸收的多余营养能量仍然残留在血液中并开始会聚到周围器官中。
Once scarring occurs, the excess nutritional energy from fat cannot be taken up by fat cells and so remains in the blood and begins to gather around organs.
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