但有关心肌缺血再灌注损伤的具体机制还未完全阐明。

But the exact mechanism of the reperfusion injury is not quite sure.

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短暂缺氧血清剥夺再灌注后，存在低能量状态，且可完全恢复。

There existed a lower energy status after transient anoxia-serum deprivation and reperfusion and the lower energy status can resume the normal level.

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自由基在继发性损伤中的具体机制尚未完全清楚，可能与缺血级联反应、缺血再灌注和细胞凋亡有关。

The mechanisms contributing to the brain damage have not yet been well clarified. The mechanisms may be related with ischemic cascade reaction, ischemic reperfusion and cell apoptosis.

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