有研究表明P 33ing1的过表达可促进由“血清饥饿”诱导的细胞凋亡，而转染反义rna则可抑制“血清饥饿”导致的细胞凋亡。
It was investigated that over-expression of P33ING1 could promote apoptosis induced by lack of serum, and transfection antisense RNA could suppress apoptosis induced by lack of serum.
结论氟可导致L - 02细胞DNA损伤，诱导细胞凋亡，适量硒可拮抗氟导致的L - 02细胞DNA损伤和细胞凋亡作用。
Conclusion fluoride exposure induces DNA damage and apoptosis of L-02 cell, and the proper level of selenium can inhibit the DNA damage and cell apoptosis induced by fluoride.
为了研究Lrp5如何影响骨特性，我们建立了骨细胞特异性表达诱导型Lrp5基因突变的小鼠，该基因突变可导致人类高和低骨量表型。
To understand how Lrp5 influences bone properties, we generated mice with osteocyte-specific expression of inducible Lrp5 mutations that cause high and low bone mass phenotypes in humans.
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