本文对7例正常人眼前房角小梁网进行了组织化学——酸性粘多糖及超微结构的研究。
Histochemical and ultrastructural studies of the trabecular meshwork(TM)of 7 normal human eyes were carried out.
结果所有患者治疗后周边前房深度均明显加深，静态前房角镜检查小梁网可见范围增宽。
ResultsThe results showed that in all these cases, the peripheral anterior chamber depth was increased, the anterior chamber angle was widened.
诱导后晚期的模型眼前房角胶原增生，结构破坏。结论复方卡波姆诱导的兔眼慢性高眼压模型房水流出受阻的主要部位在小梁内皮网部。
Conclusions the obstruction of aqueous humor outflow induced by compound Carbomer in rabbit high IOP model is caused mainly by the changes in trabecular endothelial cells.
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