结论灯盏花素可通过抑制神经细胞凋亡而减轻缺血再灌注引起的脑损伤。
CONCLUSION Breviscapine showed an improvement effect against brain impairment induced by ischemia-reperfusion in mice, and the inhibition of apoptosis in brain may be involved in.
利多卡因可减轻缺血再灌注和炎症反应所致的细胞损伤，其机制至今不明。
Lidocaine attenuates cell injury induced by ischemic-reperfusion and inflammation, although the protective mechanisms are not understood.
结论：缺血后处理可减轻缺血再灌注损伤、其机制可能与减少心肌细胞凋亡有关。
CONCLUSION: ischemic postconditioning can relieve the ischemic reperfusion injury, the mechanism of which may be related with the decreased myocardial apoptosis.
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