这其中包括钠通道基因scn5a的突变，该基因以前发现与LQTS、其他不稳定心律失常以及猝死相关。
These included mutations in the sodium channel gene SCN5A, previously associated with LQTS and other unstable heart rhythm conditions and sudden death.
由以上结果可知，SR 1耐盐性的提高是不稳定的，其耐盐性的提高并非基因突变引起。
These suggest that the salt tolerance of SR1 is not stable; its increase of salt tolerance was a physiological adaptation, not mutation.
分析表明：突变现象是感应充电进入电晕充电的固有特性，电压降低，电流突然增大，产生突变，且不稳定的起始(着火)电压与电极的结构形式、尺寸、液体介电物理性质有关。
It is found that the instable start (ignition) voltage is related to the structure and the size of electrode as well as to the physical property of liquid electric inductivity.
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