...是(1)外部非伤害性刺激因素可引发疼痛,称为异常痛(Allodynia);(2)对伤害性刺激的痛觉反应明显加剧,称为痛觉过敏(Hyperalgesia):
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...Received 2002-03-04Accepted 2002-05-29 关键词 : 痛过敏; 消炎痛; 单位放电; 糖尿病; 大鼠[gap=1610]Key words: hyperalgesia; indomethacin; unitary discharge; diabetes; rat ..
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zone of hyperalgesia 痛觉过敏区
primary hyperalgesia 痛觉过敏 ; 原发性痛觉过敏
thermal hyperalgesia 温度痛觉过敏 ; 热性痛觉过敏
secondary hyperalgesia 继发性痛觉过敏
zones of hyperalgesia 痛觉过敏带
mechanical hyperalgesia 痛觉过敏 ; 机械痛敏
auditory hyperalgesia 听觉性痛觉过敏
Types of hyperalgesia 痛敏的种类
hyperalgesia score 差值作为痛敏分数
In the inflammatory pain rat model,the previous studys showed that i. t. CRH inhibited acetic acid injection-induced hyperalgesia,and direct influence on immunological function has been discovered recently.
在炎症痛大鼠模型上,以往研究显示,鞘内注射CRH能抑制痛敏。 最近还发现CRH能直接影响免疫功能。
参考来源 - 促肾上腺皮质激素释放激素在针刺镇痛与免疫调节中的作用及机制研究Pain and hyperalgesia was caused by injury of tissue and peripheral nerves.
组织损伤及外周神经损伤后可导致痛及痛过敏。
参考来源 - 福尔马林炎性痛诱导大鼠脊髓后角和海马PKC表达上调Furthermore, astrocyte activation is sufficient to result in hyperalgesia, which can be effectively alleviated by interruption of glial including astrocytic function.
星形胶质细胞激活本身足以导致痛觉过敏,而干扰胶质细胞包括星形胶质细胞功能的药物亦能有效抑制痛觉过敏。
参考来源 - 强直刺激坐骨神经诱导脊髓痛敏神经元可塑性机制·2,447,543篇论文数据,部分数据来源于NoteExpress
Pain and hyperalgesia was caused by injury of tissue and peripheral nerves.
组织损伤及外周神经损伤后可导致痛及痛过敏。
RESULTS: Thermal hyperalgesia developed between Days 12 and 18 after cancer cell inoculation.
结果:热痛觉过敏在肿瘤细胞植入后的12 - 18天发生进展。
All these results suggested that VR1 is critical for the expression of inflammation-induced heat hyperalgesia.
这些结果进一步说明VR1在炎症性热痛敏的形成中起着非常重要的作用。
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