• 目的建立骨骼肌细胞损伤模型

    Aim To establish the injury model of skeletal muscle cells.

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  • 结论:CNTF对体外骨骼肌细胞具有营养作用

    Conclusion: CNTF is of trophic function on muscle cells in culture.

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  • 结论生理剂量地塞米松用于建立骨骼肌细胞损伤模型

    Conclusion Dexamethasone beyond physical dosage could be applied to establish injury model of skeletal muscle cells.

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  • 结论直接注射BMP2基因使其导入骨骼肌细胞表达

    Conclusion: BMP2 gene may be transfered and expressed in skeletal muscle cells by direct injection.

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  • 目的研究小鼠死后骨骼肌细胞DNA降解死亡时间关系

    Objective To study the relationship between changes of DNA degradation and the postmortem interval.

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  • 结论GH使心肌细胞骨骼肌细胞肥大不是间质组织增生

    Conclusion GH can induce hypertrophy of cardiac muscle cells and skeletal muscle cells but not interstitial proliferation.

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  • 目的完善骨骼肌细胞分离培养方法有关实验提供足量细胞

    Objective To improve the method of isolated culture of mice skeletal muscle cells and supply enough cells for some experiments concerned.

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  • 目的研究耐力运动大鼠骨骼肌细胞葡萄糖运载体4 (gt4)影响

    Objective To investigate the effect of endurance exercise on skeletal muscle cell glucose transporter 4 (GT4).

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  • 摘 要探讨不同负荷游泳训练大鼠骨骼肌细胞结构功能的影响。

    Absrtact: To investigate the structure and function changes of skeletal muscle cell membrane after different intensity endurance training.

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  • 延迟分瓶或分化培养基促使细胞分化形成骨骼肌细胞最终融合形成管。

    The cells showed high proliferative ability in the proliferative media and could form myotubes in differentiation media.

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  • 结果:注射部位局部有炎症反应部分骨骼肌细胞内可见BMP2阳性信号

    Results: Inflammatory reaction was observed in the gene-injected area and some skeletal muscle cells demonstrated BMP2 positive signals.

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  • 目的探讨抵抗素及其结合多肽RBP大鼠骨骼肌细胞摄取功能影响

    Objective To investigate the effect of resistin and resistin binding peptide(RBP) on the glucose uptake in L6 rat skeletal muscle cells.

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  • 目的探讨线粒体肌病患者骨骼肌细胞线粒体DNA基因突变情况发病机制

    Objective To study the gene mutation in mitochondrial DNA in the patients with mitochondrial encephalomyopathies, and the pathogenic mechanism of mitochondrial encephalomyopathies.

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  • 目的探讨肥胖大鼠骨骼肌细胞葡萄糖转运因子4(GLUT-4)转位变化

    Objective: To investigate the changes in the translocations in the skeletal muscle cell glucose transporter-4 (GLUT-4) in Wistar rats with obesity.

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  • 骨骼肌细胞产生活性物质可能是临床上骨骼肌转移瘤罕见性现象关键因素。

    The results revealed that skeletal muscle derived tumor suppressor(s) may play a key role in the mechanism of the rarity of metastases to skeletal muscles.

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  • 目的研究骨骼肌细胞作为基因治疗靶细胞,其合成加工分泌外源性蛋白质的功能。

    Objective: To study systematically the role of skeletal muscle cells in synthesizing, processing and secreting recombinant proteins in vitro .

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  • 动物实验结果:动物实验显示形态学观察地塞米松造成培养大鼠骨骼肌细胞损伤死亡

    The animal experiment showed that hexadecadrol resulted in the injury or death of skeletal muscle cells in rats under morphologically observation.

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  • 以上结果表明CLA动物骨骼肌细胞正常生肌分化生脂转分化都具有重要调节作用。

    All these results suggest that CLA has important regulation on normal myogenic differentiation and adipogenesis trans-differentiation of skeletal muscle cells in animals.

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  • 目的探讨罗格列酮胰岛素抵抗大鼠骨骼肌细胞葡萄糖转运蛋白4 (GLUT4)转位影响

    Objective To investigate effects of rosiglitazone on glucose transport-4 (GLUT4) protein translocation in skeletal muscle of insulin resistance rats.

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  • 目的探讨体外诱导胚间充质细胞骨骼肌细胞分化潜能,利用分化细胞构建组织工程骨骼肌模型

    AIM:To investigate the ability of dimethyl sulphoxide(DMSO) on inducing the ectomesenchymal cells to skeleton muscle cells and using these cell to construct the three-dimension model.

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  • 结论过度训练骨骼肌的损害主要是产生自由基介导的氧应激损伤,最终表现骨骼肌细胞凋亡坏死

    Conclusions Overtraining may lead to free radical-mediated injuries to the rats' skeletal muscle, cause apoptosis and necrosis as well.

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  • 结果正常大鼠骨骼肌细胞介电响应具有频率依存性,存在方向性差异,其介电弛豫现象表现两个特征频率

    Results: Thedielectric response of the skeletal muscle cells from normal rat showed dependence of frequency, discrepancy in orientation, and two characteristic frequencies.

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  • 摘要 : 本实验观察子代平方原代骨骼肌细胞胰岛素抵抗模型影响探讨改善胰岛素抵抗的作用机制

    Abstract : To observe the effect of Shizidaiping formula on insulin resistance skeletal muscle cells and to explore its mechanism in improving insulin resistance.

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  • 结果生理剂量地塞米松抑制骨骼肌细胞增殖降低其SOD水平提高MDA水平造成细胞的损伤超载

    Results Dexamethasone beyond physical dosage could suppress cells proliferation, reduce SOD level, increase MDA, and cause cells injury and Ca 2+ overloading.

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  • 目的研究不同浓度特别是高浓度体条件下骨骼肌收缩能力以及对葡萄糖跨膜转运进入骨骼肌细胞速率影响

    Objective To investigate the effects of creatine at different concentrations, especially at high contents, on muscle contraction capacity and glucose transport rate in vitro.

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  • 目的研究运动链脲佐菌素所致糖尿病大鼠骨骼肌细胞葡萄糖运载体4 (GLUT4)基因表达胰岛素受体结合的影响

    Objective: to investigate the effect of the exercise on glucose transporter4 (GLUT4) gene expression and insulin binding in streptozotocin induced diabetic rat skeletal muscle.

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  • 结果表明中等偏上的游泳负荷强度有利于减缓老龄小鼠含量的下降速度;中等偏下的游泳负荷强度则有效地减少骨骼肌细胞凋亡

    The results show that moderate to superior load swimming could slow down the bone mineral content loss but inferior load swimming could decrease skeletal muscle cells apoptosis.

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  • 目的总结细胞骨骼肌收缩功能细胞基因治疗研究中的应用。

    OBJECTIVE: To summarize the application of myoblast in skeletal muscle contraction and gene treatment research.

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  • 对该基因表达抑制引起成肌细胞过度增殖肌纤维肥大从而引起骨骼肌增生

    The inhibition of Myostatin expression can accelerate the myoblast proliferation and myofibril hypertrophy, which results in the skeletal muscle hyperplasia.

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  • 结论:成细胞可以骨骼肌收缩功能上的研究提供一个很好的研究平台基因治疗研究中的应用组织工程的研究奠定基础

    CONCLUSION: Myoblast provides a research platform for skeletal muscle contraction function, which also lays a foundation for the muscle tissue engineering research in gene treatment.

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