停搏是镉中毒的潜在性损害。
研究硒对急性镉中毒性肾损伤的保护作用。
Protective effect of selenium on acute toxic renal damage caused by cadmium was studied.
大家公认淡水生物比海水生物更容易镉中毒。
Salt-water organisms are known to be more resistant to cadmium poisoning than freshwater organisms.
目的探讨镉中毒性肾损害的细胞分子学机制。
Objective To study the cell molecular mechanism of cadmium-induced renal damage.
目的探讨微量元素对亚急性镉中毒肾损伤的保护作用。
Objective To study the protective effect of trace elements on subacute toxic renal damage induced by cadmium.
方法用氯化镉制备大鼠亚急性镉中毒模型，使其发生明显的肾损伤。
Methods Subacute cadmium poisoning model was prepared with cadmium chloride in rats to induce obvious renal damage.
目的探讨钙拮抗剂对染镉肾细胞的保护作用，为防治镉中毒提供理论依据。
Objective To explore the interferential effect of calcium antagonists on cadmium nephrotoxicity, and to provide the theory foundation in preventing cadmium toxicity.
目的探讨亚慢性镉中毒对小鼠血液系统的影响及加锌对镉中毒的拮抗作用。
Objective To study the effects of the sub chronic cadmium poisoning on haemalogical parameter changes in mice and the protective effect of zinc sulfate (ZnSO 4).
结论锌、钼对亚急性镉中毒肾损伤均有保护作用，其效果钼比锌好，这为镉中毒防治提供新的途径。
Conclusions Zinc and molybdenum might have protective effect on subacute toxic renal damage induced by cadmium. This provides a new way in prophylaxis and treatment of cadmium poisoning.
结果表明DTPA、EDTA和8102对氯化镉中毒的小鼠有明显解毒作用，尤以dtpa、EDTA解毒作用为佳。
The results indicated that DTPA, EDTA and 8102 had obvious detoxication effect after cadmium chloride intoxication, especially DTPA and EDTA.
镉是一种重金属毒物，除可造成急、慢性中毒，还有一定致畸、致癌和致突变作用。
Cadmium is a heavy metal, except it can cause acute and chronic poisoning, it also can cause teratogenic, cancer and mutation.
为评价以L-蛋氨酸预防汞、镉和铅中毒提供了化学依据。
Theyprovided a chemical evidence for evaluating the role of L-methionine in theprevention of mercury, cadmium and lead poisoning.
结果：铅镉单独染毒组和联合染毒组出现明显的中毒症状；
Result: Lead exposure group, cadmium exposure group and combined exposure group all had toxic symptom.
结果：铅镉单独染毒组和联合染毒组出现明显的中毒症状；
Result: Lead exposure group, cadmium exposure group and combined exposure group all had toxic symptom.
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