结论心房起搏频率的改变可影响房室结的有效不应期和传导功能。
Conclusion the atrial pacing rate of change can affect the AV node effective refractory period and conduction in refractory period.
它使用压电晶体将人体体动转换为电压，用此信号控制起搏频率的变化。
The model uses piezocrystal as a sensor to transfer the body mo-ving into electric signal and to control the rate.
目的研究胺碘酮对快速右心室起搏致充血性心力衰竭(CHF)犬模型心室电生理特性的频率依赖性影响。
Objective To investigate the frequency dependent effects of amiodarone on ventricular electrophysiology in right ventricular rapid pacing induced congestive heart failure (CHF) canine models.
目的探讨正常人的心脏变时能力与年龄、性别的关系，并为频率适应性起搏器的程控提供依据。
Objective To discuss the relationship between age, gender and heart chronotropic ability, and provide some data to the programming of rate adaptive pacemakers.
窦房结的这种特性可能是单传感器频率应答性起搏器不能完全符合生理需要的基本原因。
This is the reason why the rate responsive pacemaker by one sensor is not suitable for the physiologic demand completely.
不同频率心房起搏也可揭露房室结在某一频率下不能呈现的电生理现象。
Atrial pacing at different frequencies can reveal atrioventricular node can not be present at a frequency of electrophysiological phenomena.
目的应用运动负荷超声心动图评价频率适应性起搏器对心功能的影响。
Objective To investigate the effects of exercise stress echocardiography (E-Echo)on cardiac function in patients with rate-adaptive pacemakers.
方法对窦房结变时性功能障碍的16例患者，植入闭环刺激双腔频率适应性起搏器。
Methods Dual chamber rate adaptive pacemaker with closed loop stimulation were implanted into 16 patients with sinus chronotropic disability.
目的：观察合并冠心病心绞痛的病态窦房结综合征患者置入频率应答起搏器（DDDR）的临床疗效及运动耐量评价。
Objective: To observe the clinical effect and evaluate the exercise tolerance in sick sinus symptom patients with angina pectoris after implanting rate responsive pacemaker (DDDR).
起搏后TSN组维持原有的心房肌频率适应性。
起搏后TSN组维持原有的心房肌频率适应性。
The rate-dependent of TSN groups atrium was maintained after the pacing process.
结论：心脏起搏抑制房颤发生的机制可能与快频率起搏加速心房内传导、抑制了房性期前收缩、消除了窦性心动过缓和心脏长短间歇有关。
Conclusion: atrial overdrive can prevent AF, which may results from accelerating interatrial conduction, inhibiting atrial premature beat, eliminating sinus bradycardia and long short interval.
该心脏起搏器可以在体外调节起搏脉冲的宽度和频率，实现心功能损害程度不同的心衰模型的复制。
For this pacemaker, the width and rate of stimulate pulse could be adjusted outside to construct heart failure models of various cardiac impairment degrees.
并对随着基础频率心房起搏的增加房室结呈递减性传导是频率滤过作用的基础等生理现象做出解释。
With the foundation and increase the frequency of atrial pacing was reduced atrioventricular node conduction is based on the frequency filtering effect of other physiological phenomena to explain.
该起搏器以VVI模式为基础，增加舒张压的变化量对频率调节的机制。
Based on VVI mode, it's added with a mechanism of diastolic pressure change modulating pacing rate.
结论：长期高频率起搏刺激家兔左心房是建立慢性房颤模型的有效方法。
CONCLUSION: Long-term rapid left atria stimulating is an effective method in the establishment of a chronic atrial fibrillation model in rabbits.
方法对4 4例植入频率适应性起搏器的患者行运动负荷超声检查，分别于非频率适应和频率适应两种起搏状态时进行仰卧位踏车实验。
Methods E-Echo was performed in 44 patients with pacemakers. All patients underwent supine bicycle exercise tests in fixed-rate and adaptive pacing mode.
方法对4 4例植入频率适应性起搏器的患者行运动负荷超声检查，分别于非频率适应和频率适应两种起搏状态时进行仰卧位踏车实验。
Methods E-Echo was performed in 44 patients with pacemakers. All patients underwent supine bicycle exercise tests in fixed-rate and adaptive pacing mode.
应用推荐