结论含脂量38.9%的高脂饮食可诱导大鼠出现肥胖、高胰岛素血症、血脂异常等胰岛素抵抗综合征的表现。
Conclusion fat rich diet (fat content 38.9%) can induce rats the characteristic of insulin resistance syndrome such as obese, hyperinsulinemia, dyslipidemia etc.
同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat rich diet induced obesity and insulin resistance in the same species rats, the obesity prone rats had more tendency to become insulin resistance.
相比之下，EPA只是提供了部分性的保护作用预防CLA诱导的非酒精性脂肪肝病，而对胰岛素抵抗未提供保护作用。
In contrast, EPA offered only partial protection against CLA-induced non-alcoholic fatty liver disease and provided no protection against insulin resistance.
结论：高脂饲料和高果糖餐喂饲均可以诱导胰岛素抵抗大鼠模型。
CONCLUSION: high fat feed and high laevulose forage can both induce the insulin resistance in rats.
目的：观察细胞色素P 450 -CYP2J3基因对果糖诱导的高血压和胰岛素抵抗的干预作用。
AIM: to investigate the interventional effect of cytochrome P450-CYP2J3 gene on hypertension and insulin resistance induced by fructose.
目的建立高果糖餐诱导的胰岛素抵抗综合征大鼠模型。
OBJECTIVE to establish rat models with insulin resistance syndrome induced by high-fructose diet.
目的研究运动对高糖高脂饮食诱导的胰岛素抵抗大鼠脂肪细胞葡萄糖转运蛋白4（GLUT4）的影响。
Objective To investigate the effect of exercise on glucose transporter 4 (GLUT4) in adipocyte of rats with insulin resistance induced by diets.
目的：探讨葛根素对高糖高脂饮食诱导的胰岛素抵抗大鼠脂肪细胞葡萄糖转运蛋白4 (GLUT4)表达水平及其转位机制的影响。
AIM: To investigate the effects of puerarin on expression and translocation of glucose transporter 4 (GLUT4) in adipocyte of rats with insulin resistance induced by diets.
目的探讨大豆异黄酮(SIF)改善高脂高糖膳食诱导大鼠胰岛素抵抗(IR)状态的作用及可能机制。
Objective To study the effects and mechanisms of soy isoflavone (SIF) on improving insulin resistance (IR) status in IR rats induced by high-fat and high-sugar diet.
目的探讨高脂饲料诱导胰岛素抵抗(IR)动物模型的内皮功能变化及其机制。
Objective to observe the change of endothelium-dependent vasodilatation function and to explore its mechanism in insulin-resistant (IR) rats induced by high fat feed.
目的探讨解偶联蛋白在大鼠抵抗饮食诱导肥胖中的作用。
Objective To explore the role of uncoupling proteins (UCP) in resisting diet-induced obesity in rats.
除此之外，研究表明，该途径的激活是高糖诱导的胰岛素抵抗中的一个作用因素(1,2)。
Furthermore, studies suggest that increased activity of this pathway is a contributing factor to hyperglycemia-induced insulin resistance (1, 2).
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
结论：果糖可以诱导大鼠发生高血压和胰岛素抵抗，CYP2J3基因导入后可以降低果糖诱导的高血压并改善胰岛素抵抗。
CONCLUSION: fructose can induce hypertension and insulin resistance of rats. CYP2J3 gene introduction can reduce hypertension due to fructose and improve insulin resistance.
实验结束时，根据体重将高脂实验组大鼠分为饮食诱导肥胖(DIO)和饮食诱导肥胖抵抗(DIO R)大鼠，比较各组相关指标的差异。
At the end of the experiment, the high-fat diet group was divided into dietary induced obesity (DIO) and dietary induced obesity resistant (DIO-R) subgroups according to the final body weight.
在同一种系大鼠中存在饮食诱导肥胖和胰岛素抵抗的个体差异，肥胖抵抗大鼠不易发生胰岛素抵抗。
There was difference of fat - rich - diet induced obesity and insulin resistance in the same species species rats, the obesity - prone rats had more tendency to become insulin resistance.
一般认为这种情况在很大程度上是由于胃腺癌细胞对临床治疗药物诱导凋亡的抵抗。
This is believed to be largely due to resistance of gastric adenocarcinoma cells to apoptosis induced by clinically available therapeutics.
本文主要针对活性氧的产生、氧化应激诱导胰岛素抵抗和胰岛细胞功能受损等机制进行了综述。
In this paper, we summary the mechanism of the ROS generation, insulin resistance and cell dysfunction induced by oxidative stress.
目的：建立高脂膳食诱导的胰岛素抵抗大鼠模型，观察胰敏胶囊对模型大鼠发生胰岛素抵抗的阻止作用。
OBJECTIVE To observe the effect of Yimin Capsule in preventing insulin resistance in rat models induced by high lipid diet.
可见，VFB是一种良好的植物源病毒抑制剂，可诱导植物产生抗病性，增强对TMV侵染的抵抗力。
VFB could induce the host to have resistance against diseases, and strengthen its antiviral capability to tobacco mosaic tobamovirus(TMV).
目的探讨高脂喂养和脂质灌注诱导的胰岛素抵抗（IR）大鼠糖代谢、血浆抵抗素、脂联素水平和肌肉抵抗素蛋白表达的变化。
Objective To investigate the changes of glucose metabolism, plasma resistin and adiponectin in insulin-resistant (IR) rats induced by high-fat diet and lipid infusion.
目的探讨饮食诱导肥胖抵抗(DIO - R)大鼠和饮食诱导肥胖(DIO)大鼠的肥胖相关指标的变化。
Objective To study the change of obesity related index in diet-induced obesity-resistant (DIO-R) rats and diet-induced obesity (DIO) rats.
方法随机抽取正常大鼠为正常对照组，剩余大鼠使用高脂高糖饲料和注射小剂量链脲佐菌素诱导糖尿病胰岛素抵抗大鼠模型。
Methods Experimental diabetic insulin resistance was duplicated in rats by feeding high-fat and high-sugar foods plus injecting low-dose streptozotocin.
方法：建立高脂饮食诱导的肥胖、胰岛素抵抗大鼠模型，检测大鼠体重、空腹胰岛素、空腹血糖及血脂水平。
Methods the fat rat models were developed by given oil-rich food, and the body weight, fasting insulin, fasting blood sugar and blood lipid of the models were measured.
当细菌遭遇消毒剂后，Hsp33立即采取行动以保护细菌体蛋白抵抗漂白剂的诱导聚集作用。
When bacteria encounter the disinfectant, Hsp33 jumps into action to protect bacterial proteins against bleach-induced aggregation.
这些结果表明：SNP浸种预处理能提高小麦幼苗对盐胁迫的抵抗力主要是由于其缓解了盐诱导的叶片氧化损伤所致。
These results suggested that pre-soaking seeds with NO donor (SNP) increased the salt-tolerance of wheat seedlings partly due to alleviating the oxidative damage induced by salt stress.
这些结果表明：SNP浸种预处理能提高小麦幼苗对盐胁迫的抵抗力主要是由于其缓解了盐诱导的叶片氧化损伤所致。
These results suggested that pre-soaking seeds with NO donor (SNP) increased the salt-tolerance of wheat seedlings partly due to alleviating the oxidative damage induced by salt stress.
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