祛除表皮坏死细胞和使皮脂腺管畅通无阻塞，平衡油脂分泌，抑制肌肤问题。
Peel off dead epidermis cells, dredge sebum gland, balance oil secretion and prevent skin problems.
目的：探讨大疱性表皮坏死松解型药疹的临床特征及治疗效果以指导今后临床。
Objective: to analyze the clinical features and therapeutic effects on bullous epidermal necrolysis type drug eruption in order to improve clinical management of the disease.
已报道有关利妥昔单抗的毒性表皮坏死，但毒性表皮坏死和苯达莫司汀的关系尚不确定。
Toxic epidermal necrolysis has been reported for rituximab, but the relationship of toxic epidermal necrolysis to bendamustine has not been determined.
目的：探讨T淋巴细胞在中毒性表皮坏死松解症(TEN)早期角质形成细胞坏死中的作用。
Objective: to investigate the role of t lymphocytes in the pathogenesis of early keratinocyte necrosis in toxic epidermal necrolysis (TEN).
皮肤表现为表皮细胞嗜酸性坏死和基底层细胞空泡性变，表皮发生裂隙、大泡形成以及真皮淋巴细胞浸润。
The skin showed eosinophilic necrosis of epidermal cells, vacuolar degeneration of epidermal basal cells, epidermal cleft, bulla and lymphocytic infiltration of the dermis.
结论：外源野生型PTEN基因的转染能导致体外培养的高转移性黏液表皮样癌细胞变性、坏死或凋亡。
Conclusion: the exogenous PTEN gene may induce denaturalization, necrosis, and apoptosis of the highly metastatic mucoepidermoid carcinoma cells.
结论：外源野生型PTEN基因的转染能导致体外培养的高转移性黏液表皮样癌细胞变性、坏死或凋亡。
Conclusion: the exogenous PTEN gene may induce denaturalization, necrosis, and apoptosis of the highly metastatic mucoepidermoid carcinoma cells.
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