CT灌注图像能正确反应出脑缺血的存在和严重程度，可以作为评价急性脑梗塞的方法。
Ct perfusion images can reflect the presence and degree of cerebral ischemia. It is a useful tool to evaluate acute cerebral ischemia.
目的：评价大黄素抗脑缺血损伤作用，并从细胞因子水平及表达方面探讨其抑制炎性级联反应机制。
Objective: to assess emodin antagonism to cerebral ischemia injury, and to discuss the mechanism of emodin inhibiting the inflammatory cascade reaction from the levels and expressions of cytokines.
大鼠脑缺血急性期脑甲状腺激素受体向上调节为机体的重要代偿反应。
The results indicated the upward regulation of TR in rat brain during acute cerebral ischemia is a compensatory response, which allows the maximum binding of thyroid hormones.
因此，IRF-1成为在脑缺血治疗中阻断级联反应的靶点之一。
Therefore, IRF-1 has become one of the targets breaking the cascades in the treatment of cerebral ischemia.
结论颅内高压状态下进行开放式ETS可引起脑缺血、缺氧反应。
Conclusions Opening ETS can cause cerebral ischemia and hypoxia during intracranial hypertension.
目的探讨老年短暂性脑缺血发作(TIA)与C反应蛋白(crp)的关系及临床意义。
To investigate the relationship between transient ischemic attack (TIA) of the aged and serum C-reactive protein (CRP).
方法应用免疫组化方法观察大鼠局灶性脑缺血后脑组织中FAP - 1蛋白免疫反应阳性细胞数量及分布。
Methods the distribution and quantity of FAP-1 immunological positive cells in the brain were observed with immunohistochemistry method after focal ischemia.
目的观察血管紧张素转化酶抑制药卡托普利对大鼠局灶性脑缺血再灌注损伤后炎症反应的影响。
Purpose to investigate the anti-inflammatory effect of angiotensin-converting enzyme inhibitor captopril on the brain against ischemia-reperfusion injury and explore its mechanism of action.
本文探讨SOCS - 3与脑缺血后炎症反应的关系，就其在缺血性脑血管病中的研究现状作一综述。
In this article, we reviewed the current status of SOCS-3 in ischemic stroke, and focused on the relationship between SOCS-3 and inflammation after ischemic stroke.
免疫炎症反应在脑缺血后损伤中起重要作用。
The inflammatory immune response plays an important part in cerebral ischemic injury.
脑缺血后星形胶质细胞作为中枢神经系统第一个受损的细胞，出现肥大、增殖，并合成表达多种炎症介质，启动免疫级联反应。
Then ischemia stimulates hypertrophic and proliferative changes in astroglia and induces it produce inflammatory factors involved in the initiation of immunologic cascade.
皮质缺血区和尾壳核区的巢蛋白阳性反应持续到脑缺血6周。随后，其反应稍有减弱。
These alterations of nestin positive cells persisted up to 6 weeks post-ischemia, and then, the nestin positive response in the ischemic brain decreased gradually.
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral ischemic injury, etc. were reviewed and...
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral...
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral...
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