在急性缺血性肾损伤中，肾血流量是减少的。
In ischemic acute kidney injury, renal blood flow is decreased.
肝肾内皮素的升高与伤后相应肝、肾血流量减少呈明显负相关。
The changes in endothelin in liver or kidney were negatively correlated with the alteration in renal or hepatic blood flow.
测量了13例受试者基线与第6周各期肾血流量与肾小球滤过率。
In 13 subjects, renal blood flow and glomerular filtration rate were assessed at baseline and week 6 of each period.
其机理与增加肾血流量、改善肾皮质缺血区域的血液循环等因素有关。
The mechanisms of verapamil protection against ARF are:verapamil increased renal blood flow and GFR, improved renal cortical blood circulation, etc.
血管紧张素转换酶抑制剂还能增加肾血流量而通常并不改变肾小球滤过率。
ACE inhibitors also increase renal blood flow, usually without altering the glomerular filtration rate.
由于肾组织能量消耗大，肾血流量多，使其成为对缺血缺氧最敏感的器官。
Because of high consumption of oxygen and large blood flow in the renal tissues, kidney is the most sensitive organ to ischemia. So the renal injury happens as a high incidence.
慢性肾炎普通型和高血压型比肾功能正常组肾血流量显著降低（P<0.01）；
The renal plasma flow of chronic nephritis patients with hypertension and common type was more lower than that of patients with normal kidney function(P<0.01);
ARF发生时，肾脏存在异常的肾血流动力学的改变，肾血流量减少50％，肾小球有效滤过压下降。
There are inappropriate changes of renal hemodynamics that renal blood flow reduce 50%, and glomerular function reduce rapid at the process of ARF.
结论高渗盐水配合速尿能够增加心衰犬肾脏血流量，改善其降低的肾灌注，并改善部分心脏结构和功能指标和血流动力学状况。
CONCLUSION HSS plus furosemide can increase the decreased renal blood flow in canine congestive heart failure and improve kidney perfusion and some echocardiography and hemodynamic parameters.
肝素治疗组肾组织血流量明显增加。
Renal cortex blood flow was increased significantly in heparin -treated group.
肝素治疗组肾组织血流量明显增加。
Renal cortex blood flow was increased significantly in heparin -treated group.
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