本研究的目标是在MESCC已建立的模型动物中检验是否实验条件诱发的高血糖症会加速神经组织的退变。
The goal in this study was to determine whether experimentally induced hyperglycemia accelerates neurological decline in an established animal model of MESCC.
目的本研究旨在探讨运动神经损伤后运动终板退变的预防方法。
Objective To explore measures to prevent motor endplate degeneration and muscular atrophy after motor nerve injury.
在本研究中，我们试图对COX - 2和5 -LO的表达与铝过负荷致神经元退变的关系进行初步研究。方法。
In this study we try to elucidate the relationship between expression of 5-lox and COX-2 and neurodegeneration induced by aluminum overload.
当脊柱的骨头、椎间盘和关节发生退变，会形成骨刺并挤入椎管或椎间孔，这被称为椎管狭窄，也会对神经根造成有害的压迫。
When bones, discs or joints of the spine degenerate, bony spurs may form and push into the spinal canal or foramen space.
确切发病原因尚不清楚，多认为与神经纤维瘤病或脂肪瘤退变有关。
The exact pathogenesis of the deformity is still obscure, but most authors believe that macrodactyly is related to neurofibromatosis or lipomata degeneration.
越来越多的研究结果提示，COX - 2和5 - LO及其催化产物可能与神经元损伤和神经元退变有关。
More and more studies suggest that COX-2 and 5-lox, as well as their products be involved in neuron damage and neurodegeneration.
目的：观察美洛昔康对慢性铝过负荷致大鼠神经元退变的保护作用及其机制。
Objective To study the protective effects and mechanisms of meloxicam on neurodegeneration induced by chronic aluminum overload in rats.
尽管其对于中枢神经系统的作用目前并不十分清楚，大量研究表明，5lox参与了生理状态下的脑细胞增殖，并在神经元退变中扮演了重要角色。
Although its role in CNS is not completely clear, studies showed that 5lox appear to be associated with neuron proliferation in physiological status and may participate in CNS neurodegeneration.
椎间盘退变会释放炎性蛋白，刺激相邻神经，也可引起坐骨神经痛。坐骨神经痛还有许多其他的原因。
It can also occur when a disc degenerates, which releases inflammatory proteins that irritate the adjacent nerve. There are many additional causes of sciatica.
腰脊柱的退变，可能主要影响骨连结段而不是骨性段，既累及中央椎管，也累及神经根管。
The degeneration of the lumbar spine may mainly influence the articular segments other than the intraosseous segments and involve both the central vertebral canal and the nerve root canals.
目的探讨骨骼肌失神经的退变和再生。
Objective To study the degeneration and regeneration of skeletal muscle after denervation.
侧脑室微量注射氯化铝，一天1次，连续5天，建立铝负荷致小鼠神经元退变模型。
Methods(1) Neurodegenerative model of mice was established via intracerebroventricular injection of aluminum, once a day for 5 days.
本文探讨了脑源性神经营养因子、神经生长因子对体外长期培养的胚基底前脑胆碱能神经元是否具有延缓退变的作用。
The delaying effects of BDNF and NGF on the degeneration of the long term cultured embryonic basal forebrain cholinergic neurons in vitro were studied.
结论兔视神经挫伤后rgc、视神经出现渐进性退变，伴有视神经功能即刻下降。
Conclusion After the optic nerve contusion, RGCs and optic nerves showed progressive degeneration and the function of optic nerve lost immediately.
结果（1）基因工程细胞可防止多巴胺能神经元退变死亡，与对照组比较，最多增加了95 4 %，P。
Results The number of dopaminergic neurons protected by engineered cells increased at least by 95.4% in comparison with the control cells (P<0.01).
缺血后脑的病理改变以神经细胞变性、固缩和退变，皮层下白质的神经纤维和髓鞘溃变为主，普遍存在胶质细胞增生，形成胶质小结。
There were obvious pathological change in the brain after the chronic cerebral hypoperfusion, such as myelinic degeneration and formation of glial nodule in white matter.
椎间盘退变会释放炎性蛋白，刺激相邻神经，也可引起坐骨神经痛。
It can also occur when a disc degenerates, which releases inflammatory proteins that irritate the adjacent nerve.
椎间盘退变会释放炎性蛋白，刺激相邻神经，也可引起坐骨神经痛。
It can also occur when a disc degenerates, which releases inflammatory proteins that irritate the adjacent nerve.
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