结果：(1)情感障碍的癌家族发生率最高。
Results: (1) The incidence of Cancer family was highest in cases with affective disorder.
目的探讨食管癌家族史、吸烟和饮酒与食管炎症、增生、早期癌间的关联。
Objective To investigate the associations of family history of esophageal cancer, smoking and drinking to esophagitis, esophageal hyperplasia and early cancer.
目的为了探讨癌家族史阳性乳腺癌妇女er水平是否高于非癌家族性乳腺癌者。
Objective to explore whether er level of breast cancer with a positive cancerous family history shows a higher level than the breast cancer of non cancerous family.
目的：报告及分析高发癌家系的遗传方式，探讨癌家族肿瘤发生与遗传、环境及饮食习惯之间的关系。
Objective: to know the hereditary pattern of a pedigree with multi-cancer history and to explore the relationship between pathogenesis and heredity, environment and dietary pattern in cancer family.
方法：采用精神分裂症和情感障碍的家系分析法和对照比较法。结果：（1）情感障碍的癌家族发生率最高。
Method: After pedigree analysis, the prevalence rate of cancer was compared among families with Schizophrenia and with Affective disorder and normal controls.
在食管癌高发区与低发区之间、有无癌家族史之间、吸烟与不吸烟之间的血浆维生素c的水平无显著性差异。
A statistical difference in the levels was not found between individuals in the high-and low-risk areas of esophageal cancer, in cancer and no-cancer family, and of smoking and no-smoking groups.
结论：在暴露于危险因素至癌变过程中，P16基因表达缺失可能与吸烟年数、好吃肥肉、癌家族史危险因素有关。
Conclusions:In the study, we can conclude that the years of smoking, eat fat meat and family carcinoma history are the risk factors of P16 abnormal expression in CRC.
癌家族史、MIN与大肠癌发病年龄轻、右半大肠癌、大肠外癌及低分化癌的发生率高均有明显关系（P<0.01，P<0.05）；
Familial predisposition and MIN-positive re- lated strongly with early age of cancer onset, the proclivity for proximal colonic, poor differentiated and extracolorectal malignancy (P<0.01, P<0.05).
如果你有患皮肤癌的危险，尤其是有这种病的家族遗传史，一定要去咨询皮肤科医生。
If you have any risk factors for skin cancer - especially a family history of the disease - be sure to consult a dermatologist.
这意味着存在一个很强的家族史，许多血缘上有关系的亲戚患有乳腺癌或卵巢癌。
This means there is a compelling family history, including multiple blood relatives with breast and/or ovarian cancer.
拜斯博士和她的同事对78000位妇女进行了平均长达12个月的跟踪研究，她们的年龄在55岁到74岁之间，家族成员都没有卵巢癌患病史。
Dr Buys and her colleagues followed over 78, 000 women between the ages of 55 and 74 who had no family history of ovarian cancer, for an average of 12 months.
一位家族朋友说，他是死于长期对抗的胰脏癌所衍伸的问题。
A family friend said he died due to problems resulting from his long fight with pancreatic cancer.
许多研究表明黑色素瘤和其他皮肤癌具有家族性，但很难证明这种现象是基因的作用还是环境的作用。
Several studies have suggested melanoma and other skin cancers run in families, but it can be difficult to tease out the difference between the influence of genes and environment.
但是，研究者解释，癌风险和这些变异相关的程度会依个体的家族史而变化。
But, researchers explained, the extent of cancer risk associated with such mutations can vary depending on an individual's family history.
方法回顾性分析26例甲状腺髓样癌的临床资料，其中散发性24例，家族性2例。
Methods 26 cases of MTC underwent surgical treatment were retrospectively reviewed, including sporadic in 24 cases and familiar in 2 cases.
结论：不孕和孕次较少、动物脂肪摄入量多和有肿瘤家族史是卵巢癌发生的主要危险因素。
Conclusions: The nulliparity or low parity, high fat diet and family history of cancer were major risk factors of ovarian carcinoma.
诊断时家族性卵巢癌与散发性卵巢癌分期无差别。
Stage at diagnosis had no difference between familial and sporadic ovarian cancer.
卵巢癌的家族病史是最重要的危险因素，5 - 10%的病例存在着家族病史。
A family history of the illness is the most important risk factor and occurs in 5-10 percent of cases. The illness is influenced by hormones.
“另外，女性也应该多与家人沟通，以了解所在家族中是否有人有过乳腺癌或卵巢癌的病史。” ”
'Women also need to talk to their family to find out if there is a history of breast and ovarian cancer. '
家族病史中有人获得皮肤癌的也更容易获得皮肤癌。
髓样癌可以是散在单发，也可以是家族性发病。
联合检测SFRP基因家族甲基化状态对于食管癌的预后判断有一定指导意义。
Combination analysis of methylation status in SFRP genes may has definite value on estimating prognosis of ESCC.
鼻咽癌的发生是多种病因通过多种途径共同参与的过程，其中包括EB病毒感染、地域环境因素影响、饮食习惯以及家族遗传等。
The etiology of nasopharyngeal carcinoma (NPC) is multi-factorial, including latent infection of Epstein-Barr Virus (EBV), environmental factors, dietary habits, familial heredity etc.
鼻咽癌的发生是多种病因通过多种途径共同参与的过程，其中包括EB病毒感染、地域环境因素影响、饮食习惯以及家族遗传等。
The etiology of nasopharyngeal carcinoma (NPC) is multi-factorial, including latent infection of Epstein-Barr Virus (EBV), environmental factors, dietary habits, familial heredity etc.
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