激素敏感脂肪酶(HSL)是动物体脂肪分解的关键酶。
Hormone sensitive lipase (HSL) is a key enzyme in fat metabolism.
激素敏感脂肪酶（HSL）基因影响着脂肪的沉积，它在表达或活性上的轻微变异均有可能影响猪的背膘厚和瘦肉率。
The gene affects the accumulation of lipid and its small variation on the activity or gene expression can influence the lean percentage and backfat traits.
目的探讨高脂饮食诱导肥胖易感(OP)大鼠和肥胖抵抗(OR)大鼠，激素敏感性脂肪酶(HSL)和脂蛋白脂肪酶(LPL)基因表达的差别。
Objective To explore the gene expression of hormone sensitive lipase (HSL) and lipoprotein lipase (LPL) between obesity-prone (op) and obesity-resistant (or) rats induced by a high-fat diet.
已有证据显示PKA通过磷酸化脂周素调节脂解（13-17），引起构象改变使脂滴暴露给内源性的，激素敏感的脂肪酶（14）。
Evidence suggests that PKA regulates lipolysis by phosphorylating perilipin (13-17), resulting in a conformational change that exposes lipid droplets to endogenous, hormone-sensitive lipases (14).
已有证据显示PKA通过磷酸化脂周素调节脂解（13-17），引起构象改变使脂滴暴露给内源性的，激素敏感的脂肪酶（14）。
Evidence suggests that PKA regulates lipolysis by phosphorylating perilipin (13-17), resulting in a conformational change that exposes lipid droplets to endogenous, hormone-sensitive lipases (14).
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