一升茶树含有高达9毫克的氟化物，过量会导致氟骨症，降低骨骼质量，导致疼痛和韧带僵硬。
A litre of teacan contain up to 9mg of fluoride, which in excess can cause skeletal fluorosis, reducing bone quality and causing pain and stiffening of the ligaments.
目的：复制不同类型燃煤型氟骨症动物模型。
Objective:To establish the animal model of skeletal fluorosis caused by coal-burning.
目的研究复方硼砂片治疗氟骨症的机理及疗效。
Objective To know mechanism and efficacy of compound borax tablet in treatment of skeletal fluorosis.
仅在中国，估计就有1000多万人患有氟骨症。
In China alone, more than 10 million people are estimated to suffer from skeletal fluorosis.
海边的典型氟骨症病人全血、尿元素均显著增高。
The element content in blood and urine increased markedly for the typical fluorosis bone patients by the seaside.
氟骨症患者的治疗是地方性氟中毒防治的重要内容。
We regard treatment of skeletal fluorosis as an important content for controlling of endemic fluoride intoxication.
目的了解氟骨症发生的分子机理的研究现状和发展趋势。
Objective To explore the current research and the developing trend on molecular mechanism of endemic fluorosis skeletal fluorosis.
氟骨症是由于摄入过多的氟化物而引骨关节损害性疾病。
Objective Skeletal fluorosis is known as joint disease which is caused by excessive intake of fluoride.
氟骨症患者尿氟与骨矿物质代谢以及胶原降解有一定的关系。
There is relationship in the catabolism of bone mineral, bone collagen and bone fluoride.
目的 了解改水降氟18年后氟骨症患者机体的功能代谢状况。
Objective To analyze the functional metabolism changes of drinking-skeletal fluorosis patients after water improvement for 18 years.
结果骨形成相关基因和骨吸收相关基因与氟骨症的发生密切相关。
Results There was close relationship between the development of skeletal fluorosis and bone formation related gene and bone resorption related gene.
结论全椎板减压术是治疗氟骨症性胸椎管狭窄症有效的手术方式。
Conclusions Omni-posterior decompression procedure is proved to be an effective way to treat thoracic spinal stenosis due to osteofluorosis.
结论氟骨症患者的病情与从饮砖茶中氟摄入量呈正相关剂量反应关系。
Conclusion There was a positive correlative dose-response relationship between skeletal fluorosis state and amount of brick tea intake.
结论X线骨盆及其骨小梁改变分别是氟骨症检查的首选部位和可靠指标。
Conclusions X-ray change of pelvis and bony trabecula are selected as reliable index for diagnosis of sclerotic fluorosis.
结论X线骨盆及其骨小梁改变分别是氟骨症检查的首选部位和可靠指标。
X-ray change of pelvis and bony trabecula are selected as reliable index for diagnosis of sclerotic fluorosis.
结果氟骨症患者组与病区对照组间VMA、HVA含量有非常显著差异。
Result the content of VMA and FfVA between the fluoride group and control group is the significant difference.
其结果是，许多人患有各种不同病症，轻则牙氟中毒，严重者致残性氟骨症。
As a result, many suffer conditions ranging from mild dental fluorosis to crippling skeletal fluorosis.
探讨饮水氟浓度与人群骨关节炎患病的关系，为防治地方性氟骨症提供科学依据。
To explore the relations between fluorine concentration in drinking water and osteoarthritis, and to provide evidence for prevention and intervetion of endemic fluoride of bone.
结论：过量氟造成骨转换增高，破骨性骨吸收增强，低钙偏食是氟骨症的促发因素。
Conclusion The results prove that excessive fluoride can induce accelerative bone turnover and active bone resorption, and low calcium diet is an important promotive factor of skeletal fluorosis.
目的观察饮水型与燃煤污染型两类地方性氟中毒病区氟骨症骨关节损害的放射学差异。
Objective Observing the radiology difference for bone joint of skeletal fluorosis between drinking water of Jilin and burning coal area.
结论磷肥生产中氟化物对作业工人上呼吸道，骨骼和牙齿有损害作用，可导致氟骨症。
The fluoride in phosphate fertilizer production can not only impair workers' upper respiratory tract, bone and teeth, but also lead to fluorosis of bone.
结果表明，氟骨症组血清T_4低于对照组，而血清TSH高于对照组，血清T_3无明显改变。
The results showed that serum T_4 in the patients was lower than in the controls and TSH was higher.
低浓度对牙齿有益，但浓度过高则可能导致使人衰弱的疾病，比如氟骨症，一些社区因此而成为一片废墟。
Low concentrations are good for teeth, but excessive concentrations can lead to debilitating disease, such as skeletal fluorosis, which has devastated some communities.
目的：观察中度地方性氟骨症患者骨骼损害的X线表现特点以及郭氏中药疗法治疗前后X线的变化情况。
Objective: To observe the X-ray features of bone damage in patients with moderate endemic skeletal fluorosis and the changes of X-ray after treatment with herbal therapy.
结论氟中毒初期就有神经系统改变，尿中儿茶酚胺酸性代谢物的测定对氟骨症早期诊断及治疗有积极的意义。
Conclusion There is the change of nervures system during early period of Fluoride. It is an active significant that measuring the ca metabolite can make an early diagnosis...
结论深入研究氟骨症的分子发病机制，尤其是小儿氟骨症的分子发病机制，对于制定氟骨症的早期预防和控制措施具有重要的意义。
Conclusion It may play an important role in the early prophylaxis and control measures studying the molecular mechanism of endemic fluorosis skeletal fluorosis especially in children.
结论深入研究氟骨症的分子发病机制，尤其是小儿氟骨症的分子发病机制，对于制定氟骨症的早期预防和控制措施具有重要的意义。
Conclusion It may play an important role in the early prophylaxis and control measures studying the molecular mechanism of endemic fluorosis skeletal fluorosis especially in children.
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