本品是拟除虫菊酯类杀虫剂。
达到了拟除虫菊酯类农药残留的分析要求。
It reaches the requirements of fenpropathrin residues analysis.
目的:了解拟除虫菊酯类制剂杀灭蟑螂的效果;
Objective:To know the killing effect of pyrethroid preparation against cockroach;
建立家兔拟除虫菊酯类农药灌胃染毒致死模型。
To develop a pyrethroid intragastric administration death model.
电压敏感的钠通道是DDT和拟除虫菊酯类杀虫剂的主要靶标。
The voltage-sensitive sodium channels are important action targets of DDT and pyrethroids.
方法采用幼虫浸渍法,测定其对拟除虫菊酯类杀虫剂的抗性倍数。
Methods the larvae dipping method was used to test the resistance to pyrethroid.
报道了一种新型结构的拟除虫菊酯类似物的合成以及杀虫活性试验。
A new chemical structure category of pyrethroids was synthesized. The tests in insecticidal activity was reported.
该方法快速、准确、可靠,适合于测定水产品中拟除虫菊酯类药物的残留。
This method can detect pyrethroids residues in aquatic product quickly and exactly.
研究了采用微波辅助萃取法从中药材中提取拟除虫菊酯类农药的影响因素。
A method to determine pyrethroid residues in traditional Chinese medicines based on microwave-assisted extraction was studied.
探索II -型拟除虫菊酯类农药对小鼠脑的氧化损伤作用及其毒理作用机制。
To study oxidative damage of II-Pyrethrins on brains of mice and the mechanism of its toxic effect.
S101与有机磷、拟除虫菊酯类杀虫剂复配防治害虫有增效作用,对动物不增毒。
The combined application of S101 and organophate or pyrethroid insecticides had obvious synergism to pest insects, its toxicity to mammalian was not increased.
毒理学和电生理学的研究表明,在许多拟除虫菊酯类杀虫剂抗性昆虫中存在击倒抗性。
Toxicological and electrophysiological studies have demonstrated the presence of Kdr-type resistance in many pyrethroid-resistant insects.
拟除虫菊酯类杀虫剂对昆虫的选择毒性很可能是因为昆虫神经元的钠通道结构与哺乳动物的不同。
The selective toxicity of pyrethroids for insects probably resulted from configuration differences of the sodium ion channels in nerve cells between insects and mammals.
拟除虫菊酯类杀虫剂对昆虫的选择毒性很可能是因为昆虫神经元的钠通道结构与哺乳动物的不同。
The selective toxicity of pyrethroids for insects probably resulted from configuration differences of the sodium ion channels in nerve cells between insects and mammals.
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