• 最近动物研究中,这种化学物质抑制了COX - 2(环氧化)——引发炎症一种蛋白质(相同的蛋白质称为COX - 2抑制剂药物西乐葆则则无效)。

    In recent animal studies, this chemical inhibited COX-2, a protein that spurs inflammation (the same protein that so-called COX-2 inhibitor drugs such as Celebrex quash).

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  • 治疗过程中的步骤需要一种,这种能促发新陈代谢以及信号转换从而引发皮肤修复重塑进程

    One step in the healing process involves an enzyme that starts a metabolic effect and a signal transduction that triggers the process of skin repair and skin remodeling.

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  • 这些反应引发通过第二信使被连接在一起。

    These reactions are carried out by enzymes and linked through second messengers.

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  • 蛋白一种蛋白质,对于引发AIDSHIV病毒生命周期至关重要

    Protease is a protein crucial to the life cycle of HIV, the AIDS-causing virus.

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  • 起因脂肪分解产物由于细菌的作用皮脂腺分泌物的潴留而渗入周围组织从而引发肉芽肿性炎症反应

    They develop when lipid breakdown products leak into the surrounding tissues from either bacterial enzymes or retained sebaceous secretions and incite a granulomatous inflammatory reaction.

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  • 英国内科医生。他首先发现一类新的基因诱导疾病是由于某一个基因发生突变从而导致某种特殊缺乏产生故障而引发的。

    British physician who first recognized a new class of genetically induced diseases resulting from a mutation in a single gene that causes the absence or malfunctioning of a particular enzyme.

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  • 感染引发早产机制与前列腺合成细胞因子的异常分泌基质金属蛋白活性增加等有关。

    The mechanism of premature delivery caused by infection involves in increased prostaglandin synthesis, abnormal secretion of cytokines and increased activity of matrix metalloproteinases.

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  • 丙酮氧化能为催化琥珀酸所引发,说明有缩合的活性存在。

    Pyruvate oxidation was accelerated by catalytic amount of succinate. The operation of condensing enzyme is thus indicated.

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  • 因此肾脏组织型血管增渗可能被败血症引发发炎反应刺激释放出来产生作用。

    Therefore, it is possible that renal tissue kallikrein-bradykinin system is also released and activated by the stimulation of inflammation in sepsis.

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  • 这种可能有助于降解信息素,并且增加了有关发现对手能力能够引发好斗行为理论的证据

    The enzyme may also help break down pheromones, adding to evidence that an ability to sniff out rivals can trigger aggressive behaviour.

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  • 他们发现正常细胞中,BCL9L帮助激活一种叫做半胱天冬-2的蛋白质应对异常数量染色体引发程序

    They revealed that in normal cells, BCL9L helps activate a protein called caspase-2 in response to an abnormal number of chromosomes, triggering a self-destruct sequence.

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  • 即便诱使其他细胞产生引发新的问题复制越多变异可能性越大细胞便会癌变

    And even if we could trick the rest into producing it, then we'd have another problem: more replications means more chances for mutations that could turn a cell cancerous.

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  • 即便诱使其他细胞产生引发新的问题复制越多变异可能性越大细胞便会癌变

    And even if we could trick the rest into producing it, then we'd have another problem: more replications means more chances for mutations that could turn a cell cancerous.

    youdao

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