• 目的观察大鼠灶性缺血失联络现象及中药川芎嗪的干预作用

    The objective is to observe the crossed cerebellum diaschisis after focal cerebral ischemia and the interventional effects of ligustrazine.

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  • 局灶性脑缺血后亦可增加海马齿状神经发生。

    Focal ischemia also could raise the neurogenesis in hippocampus dentate gyrus.

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  • 目的研究性脑缺血-再灌注损伤模型

    Objective: To study the model of the focal cerebral ischemia-reperfusion injury in rats.

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  • 目的研究复灵局灶脑缺血保护作用

    Objective: To investigate the protective effects of NFL on local cerebral ischemia in rats.

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  • 方法阻断一侧动脉造成局灶缺血模型

    Methods the model of cerebral ischemia was established by the unilateral middle cerebral artery occlusion in the rats.

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  • 目的探讨电针对恢复期性脑缺血大鼠治疗作用

    Objective To observe the effects of EA on recovery of focal cerebral ischemia in rats.

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  • 通过经颅阻断大鼠动脉制成缺血动物模型

    A model of cerebral infarction was established by occluding the left middle cerebral artery (MCA) transcranially.

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  • 结论对大鼠局灶性缺血-再灌注损伤保护作用。

    Conclusion: Qileng Decoction can protect the brain cerebral ischemia reperfusion injury.

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  • 结论局灶性脑缺血引发NMDA受体迅速持续升高

    Conclusions Focal cerebral ischemia can result in prompt and continuous increase of NMDA receptor activity.

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  • 目的:研究针刺大鼠局灶性缺血血清影响

    Objective: to approach to the effects of acupuncture on serum Calcium, Magnesium and Phosphours of rats with acute cerebral ischemia.

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  • 目的探讨阿司匹林局灶脑缺血保护作用及其机制

    AIM To investigate the protective effects of aspirin on local cerebral ischemia in rats and its mechanism of action.

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  • 前言:目的:研究中风康大鼠灶性脑缺血再灌注损伤影响

    Objective: to study the pharmacological effects of Zhong Feng Kang on model mice with cerebral ischemia and after restoration of blood flow.

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  • 针刺血清调节可能针刺治疗局灶性脑缺血机理之一

    The effect of serum calcium, magnesium is one of the mechanism of acupuncture therapy on acute cerebral ischemia.

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  • 关于绿茶多酚大鼠性脑缺血缺血区BBB作用见报道

    However, the effects of green tea polyphenols on BBB of focal cerebral ischemia rat has not been reported.

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  • 结论局灶性脑缺血损伤有白细胞流变学异常变形能力下降

    CONCLUSION: the acute focal cerebral ischemic injury leads to the abnormal leukocyte rheology and declining of deformability.

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  • 目的本文旨在研究性脑缺血,大nmda受体变化。

    Objective to study the alteration of NMDA receptor activity during focal cerebral ischemia injury in rats.

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  • 目的观察大鼠灶性缺血失联络现象及中药川芎嗪的干预作用

    AIM: To observe the crossed cerebellum diaschisis after focal cerebral ischemia and the interventional effects of ligustrazine.

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  • 电针可以显著提高性脑缺血大鼠血清SOD降低血清mda含量

    Electroacupuncture can improve serum SOD activation and reduce MDA content notably of focal cerebral ischemia rats.

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  • 结论附注射液对大鼠短暂性脑缺血损伤呈非剂量依赖保护作用

    Conclusions:The Shenfu injection possesses dosageindependent protective effects on transient focal cerebral ischemic injury.

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  • 目的研究性脑缺血调控蛋白(GRP78GRP94)表达

    Objective To investigate the expression of the glucose regulated protein (GRP78, GRP94) in rats during focal cerebral ischemia.

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  • 目的研究短暂局灶缺血预处理对再次缺血微血管损伤水肿保护作用。

    Objective To investigate the protection of brief focal cerebral ischemic preconditioning against microvascular injury and brain edema after subsequent severe focal ischemia.

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  • 目的观察银杏提取物GBE大鼠局灶性缺血保护作用探讨作用机制

    Objective To observe the effect of Ginkgo biloba extract(GBE) on cerebral ischemia injury in rats and explore its mechanism.

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  • 目的采用MRI检测神经功能缺失评分判定线栓法制作大鼠局灶缺血动物模型可靠

    Objective To detect the reliability of focal cerebral ischemia model which is evaluated by the score standard of neural function deficient degree after using MRI.

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  • 结论DG明显抑制灶性缺血-再灌注所诱导神经细胞凋亡,起到一定的神经保护作用。

    Conclusion DG can markedly inhibit nerve cell apoptosis induced by focal brain ischemia reperfusion injury and protect the nerve cell to a certain extent.

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  • 建立一种符合临床缺血发病规律的动物模型是研究性脑缺血发生机制防治措施基本条件

    To establish the animal models similar to the clinical condition is the foundation for researching the pathogenesis and therapeutic strategies about focal cerebral ischemia.

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  • 为了研究针刺大鼠局灶性脑缺血血清含量影响,对针刺作用机理做进一步的探讨。

    To approach to the effect of acupuncture on serum calcium, magnesium in the rats of acute cerebral ischemia.

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  • 结论局灶性脑缺血后,调节NO生成量适当范围可以达到保护防止神经细胞进一步损伤目的

    Conclusion in the process of focal cerebral ischemia and reperfusion, regulating the quantity of NO in the propriety range may protect and avoid purpose of neuronal cell injury.

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  • 目的研究血红素氧合酶1 (HO 1)血红素氧合酶2 (HO 2)局灶性脑缺血中的作用。

    Objective To study the different role of heme oxygenase-1 (HO-1) and heme oxygenase-2 (HO-2) protein during permanent focal cerebral ischemia in rats.

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  • 结论透光结合神经症状综合评分用于小鼠局灶性脑缺血定量分析药物的神经保护作用评价

    CONCLUSION: Light transmission and integrated graded approach can be used not only for qualitative analysis of focal cerebral ischemia, but also for evaluating the neuroprotective effect of drugs.

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  • 结果血栓片可依剂量减轻局灶缺血所致梗塞体积以及改善脑缺血引起的神经症状

    Results KNT could alleviate volume of cerebral infarct caused by focal cerebral ischemia in a dose- dependent manner and improve neurological symptoms.

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