容量血管收缩，静脉回流量增加。
Capacitance vessels constrict, increasing the back flow of venous blood.
其作用源于大的容量血管和小的阻力血管的同时扩张。
This effect is due to dilation of both the large conductance and small resistance renal blood vessels.
结论HD成功的关键在于血管通路的建立、血容量的稳定及并发症的防治。
Conclusions: The key factory of HD success is to construct vascular passage, establish blood volume and prevent complication.
妊娠期间，血容量和心输出量增加30- 50%，而全身血管阻力下降。
During pregnancy, plasma volume and cardiac output increase by 30-50%, while systemic vascular resistance is reduced.
在肾素-血管紧张素-醛固酮系统中，肾小球旁体帮助调节血容量和压力。
In the renin - angiotensin - aldosterone system, the juxtaglomerular apparatus helps regulate volume and pressure.
过度利尿可能会导致脱水和减少在血容量，与循环系统崩溃的可能性与血管血栓和栓塞，特别是在老年患者。
Excessive diuresis may result in dehydration and reduction in blood volume, with circulatory collapse and with the possibility of vascular thrombosis and embolism, particularly in elderly patients.
休克型以扩充血容量，纠正酸中毒基础上加用血管活性药物。
The treatment of shock type is based on the rectification of acid intoxication and use vascular active medicine to enlarge the blood capacity.
肺血管是压力反射效应器的一部分，立位应激时的压力反射很大程度上控制着肺循环的阻力和容量。
Pulmonary vascular is an effector of baroreceptor reflex, which can exert significant control of pulmonary resistance and capacitance after orthostatic challenge.
快速给予抑制剂的缺点是心血管功能有问题或循环容量在临界水平的病人可发生低血压。
The disadvantage of giving depressant drugs rapidly is that hypotension may occur in patients with questionable cardiovascular status or marginal circulatory volume.
水肿和血管内容量。
肾素-血管紧张素-醛固酮系统（RAAS）是一种能被容量丢失激活的反馈控制系统。
The renin-angiotensin-aldosterone system (RAAS) is a feedback system activated with volume depletion.
血管紧张素转换酶抑制剂通过舒张血管和减少血容量而降血压。
They reduce blood pressure through vasodilation and reduction of blood volume.
干涉：血管痉挛通过血管造影和经颅多普勒超声证实，针对血容量过多、高血压和血液稀释等症状进行治疗。
INTERVENTION: Vasospasm was confirmed by angiography and transcranial Doppler sonography, and symptoms responded to hypervolemia, hypertension, and hemodilution therapy.
肾素-血管紧张素系统作用机制的第一步是肾近球细胞受到低血压、交感神经兴奋、低血容量及低钠流量的刺激而分泌肾素。
The first step in the RAS cascade is the secretion of the enzyme renin by renal juxtaglomerular cells in response to low blood pressure, sympathetic activation, hypovolemia, and low sodium flux.
分布性休克： 分布性休克源于因动脉或静脉扩张引起的血管内容量的相对不足，其循环血量正常。
Distributive shock: Distributive shock results from a relative inadequacy of intravascular volume caused by arterial or venous vasodilation;
分布性休克： 分布性休克源于因动脉或静脉扩张引起的血管内容量的相对不足，其循环血量正常。
Distributive shock: Distributive shock results from a relative inadequacy of intravascular volume caused by arterial or venous vasodilation;
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