目的探讨妊高征的临床特点。
RAS基因是妊高征主要的致病基因。
妊高征经早期临床干预后可以降低其发病率。
The incidence rate of pregnancy induced hypertension can be decreased by earlier period clinical intervention.
目的分析妊高征患者蜕膜粘附分子表达的变化。
Objective To analyze the levels of cell adhesion molecules on deciduas from patients with pregnancy induced hypertension.
所以于妊娠中期补钙可以降低妊高征的发病率。
Therefore complementing calcium in midterm pregnancy can reduce the morbidity of hypertension of pregnancy.
目的研究茵陈汤是否具有预防及治疗妊高征的作用。
Objective to study tarragon soup whether has the role to prevent and cure pregnancy induced hypertension syndrome (PIH).
结果妊高征胎儿脐动脉血流阻力指标不同程度增高;
Results The index of the PIH fetal umbilical arterial bloodstream's asistance increases more or less.
结果导致MOF的主要因素为重度妊高征及产后出血。
Results Severe PIH and postpartum hemorrhage were the major factors that led to MOF.
妇女对妊高征的知识得分中位数为1分(满分6分)。
The median score of women's knowledge about pregnancy-induced hypertension is only 1 (total is 6).
目的:观察硝苯地平对妊高征患者眼动脉血流的影响。
Objective: To evaluate the effect of nifedipine on ophthalmic artery blood flow patients with pregnancy induced hypertension (PIH).
结论妊娠心衰的主要病因为先心、风心和严重妊高征;
Conclusion The main causes of heart failure in pregnancy are congenital and rhumatic heart diseases and PIH.
结果妊高征及慢性高血压病占本组病因的46。 2%。
Results Pregnancy-induced hypertension syndrome and chronic hypertension were the main pathogeny of this disease(46.2%).
妊高征组胎盘滋养层细胞超微结构有明显的病理学改变。
The obvious pathological changes were found in ultrastructure of placental trophoblast of study group.
目的:测定妊高征患者血清睾酮水平并探讨其临床意义。
Objective: to determine maternal serum testosterone levels in patients with pregnancy - induced hypertension (PIH) and evaluate its clinical utility.
并发症中以妊高征为最多,其次为贫血、产后出血和早产。
Gestation hypertension was the most in the complications, then was anemia, postpartum hemorrhage and premature delivery.
妊高征患者胎盘床蜕膜t淋巴细胞亚群的表达与临床意义。
The expression of T-lymphocyte subpopulations of decidual-placental bed in pregnancy with pre-eclampsia and clinical significance.
观察了220例中、重度妊高征患者的眼底情况并进行分析。
Methods The fundus of 220 patients with moderate and severe PIH were examined and analyzed.
目的:探讨血小板活化、凝血激活与妊高征的关系及临床意义。
Objective: To discuss the clinical significance of platelet activation, coagulation activation and pregnancy-induced hypertension syndrome.
HO可能在妊高征患者胎盘组织损伤的病理生理中起重要作用。
Ho may play a role in the pathophysiology of poor placental perfusion and tissue damage in placenta of PIH.
目的:探讨海平与高原地区妊高征患者血液纤溶状态的变化情况。
Objective: To investigate the changes of the fibrinolysis coagulation system in patients with pregnancy induced hypertension(PIH)at sea level and highland.
目的:研究妊高征患者血浆纤维蛋白原单体聚合功能的临床意义。
Objective: To study the changes and the clinical significance of Plasma fibrinogen monomer polymerization function in PIH.
结论:连续脊神经阻滞对妊高征大鼠的蛋白尿具有明显改善作用。
CONCLUSION: Continuous spinal nerve block can obviously improve proteinuria in rats with PIH.
结论胎盘缺氧是导致妊高征妇女血清瘦素水平明显升高的主要原因。
Conclusion Placental hypoxia is the major factor resulting in the increase in the serum leptin level in PIH women.
结论:妊高征阻碍胎儿发育进展随程度而加重,主要累及肺、肝等脏器。
Conclusions: The PIH syndrome had retarded the process of fetal growth and development, and associated with the severity of PIH, mostly involving the lung and the liver.
结论:剖宫产、脐带异常、早产、妊高征是导致新生儿窒息的主要原因。
Conclusion: The major causes of neonatal asphyxia were cesarean section, abnormal cord, premature delivery, and PIH.
目的:探讨人类白细胞抗原系统(HLA)与妊高征(PIH)的关联。
Objective: To explore the mechanism of correlation between human leucocyte antigen (HLA) and pregnancy induced hypertension (PIH).
结果:98例妊高征中有34例围产儿预后良好,64例围产儿预后不良。
Results: the perinatal outcomes of 34 out of these 98 cases were good, 64 were poor.
结果胎膜早破,胎儿宫内窘迫,重度妊高征,前置胎盘是引起早产的主要因素。
Premature rupture of membrane, fetal distress, severe pregnancy induced hypertension, placenta praevia were the main risk factors of premature delivery.
目的通过对孕妇胫后动脉血流指数的检测,为妊高征预测性诊断提供一条新途径。
Objective To provide a new method for the predictive diagnosis of PIH through the detection of the PTA blood flowing indexes of the pregnant women.
围产儿预后不良发生率,中、重度妊高征明显高于轻度妊高征(P<0.01)。
There was a higher incidence of poor perinatal outcomes in severe and moderate PIH than in mild cases(P< 0. 01).
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