• 在治疗的第3年,雌激素受体阳性患者获得97%的无远处复发生存率,绝对危险度大幅下降11%。

    At 3 years, ER-positive patients achieved a DRFS of 97% and a significant absolute risk reduction of 11%.

    youdao

  • 目的:观察胰岛素受体基因第17外显子基因多态性与中国人脑梗死和原发性高血压的相关性。

    AIM: to observe the correlation of the 17 exon gene polymorphism of insulin receptor gene and the Chinese cerebral infarction and primary hypertension.

    youdao

  • 目的:观察胰岛素受体基因第17外显子基因多态性与中国人短暂性脑缺血发作和原发性高血压的关联。

    AIM: to observe the association of insulin receptor gene exon 17 polymorphism with the transient ischemia attack and essential hypertension in Chinese.

    youdao

  • 目的探讨中国人非小细胞肺癌表皮生长因子受体(EGFR)第18、19、21外显子基因突变状态。

    Objective to study the mutation patterns of epithelial growth factor receptor (EGFR) exon 18, 19 and 21 in Chinese non-small-cell lung cancers (NSCLC).

    youdao

  • 第1个模型是通过注射了血管内皮生长因子的抗体,或者可溶性的血管内皮生长因子受体1,从而获得的。

    The first model is generated by injection of antibodies to vascular endothelial growth factor or soluble vascular endothelial growth factor receptor 1.

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  • 目的:观察第3 ~ 8周人胚中肾的发育和生长因子及其受体的表达。

    Objective: to observe the development of mesonephros and the expression of growth factors and their receptors in human embryo of 3th-8th weeks.

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  • 结论单纯型肥胖儿童瘦素受体基因第20外显子存在基因多态性的变化,且这种变化明显影响肥胖儿童的脂质代谢及体脂分布。

    Conclusion There is polymorphism at 20 exon LEPR gene in children with obesity, which may affect the lipid metabolism and the fat distribution.

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  • 方法采用聚合酶链式反应-单链构象多态性方法检测46例高血压患者和39例正常血压对照者的胰岛素受体基因第17、18外显子的多态性。

    Methods The polymorphism if exon 17 and 18 of insulin gene were detected in 46 hypertension patients and 39 normal controls using PCR and single strand conformation polymorphism.

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  • 第2阶段关于T - bomb II它的“雌激素袭击”使用雌激素阻挡混合物去阻碍受体,确保雌激素不会依附在受体以及释放任何雌激素效用。

    Stage 2 of T-BOMB it's "estrogen assault" USES estrogen-blocking compounds to clog the receptors, assuring that estrogen does not attach to the receptor and exert any of its estrogenic effects.

    youdao

  • 第2阶段关于T - bomb II它的“雌激素袭击”使用雌激素阻挡混合物去阻碍受体,确保雌激素不会依附在受体以及释放任何雌激素效用。

    Stage 2 of T-BOMB it's "estrogen assault" USES estrogen-blocking compounds to clog the receptors, assuring that estrogen does not attach to the receptor and exert any of its estrogenic effects.

    youdao

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