目的研究前炎症细胞因子对人胸膜间皮细胞（HPMC）释放纤溶酶原激活物抑制剂（PAI1）的诱导效应。
Objective To investigate effects of two proinflammatory cytokines on the production of plasminogen activator inhibitor 1(PAI 1) by human pleural mesothelial cells(HPMC).
纤溶酶原和尿激酶前体的相互活化作用在纤溶的开始和加速上发挥着重要的作用。
Reciprocal activation of prourokinase and plasminogen is an important mechanism in the initiation and propagation of local fibrinolytic activity.
在两个试验中，在组织纤维蛋白溶酶原激活剂使用前，都先用NIHSS评分。
NIHSS scores were determined before tissue plasminogen activator bolus and at 60 and 120 minutes in both data sets.
结果在狗后肢连续应用止血带6小时，应用或解除止血带时，实验侧肢体静脉血凝血酶原时间、凝血酶时间均比应用前明显延长（P< 0 .0 5 ）。
Results Tourniguet was applied on the hind leg of the dog for 6h, thrombinogen and thrombin time were significantly longer than those found in the control group(P<0.05).
结果在狗后肢连续应用止血带6小时，应用或解除止血带时，实验侧肢体静脉血凝血酶原时间、凝血酶时间均比应用前明显延长（P< 0 .0 5 ）。
Results Tourniguet was applied on the hind leg of the dog for 6h, thrombinogen and thrombin time were significantly longer than those found in the control group(P<0.05).
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