脂肪形成是前脂肪细胞分化成脂肪细胞的过程。
Adipogenesis is the cellular process where preadipocytes differentiate into adipocytes.
目的探讨马钱苷对大鼠前脂肪细胞增殖与分化的影响。
Effects of bFGF on preadipocytes on proliferation and differation in vitro;
研究结果提示抵抗素对猪前脂肪细胞生长具有双重作用。
The results indicated that the resistin has dual effects on growth of porcine preadipocyte.
目的探讨六味地黄丸对大鼠前脂肪细胞增殖与分化的影响。
Objective To investigate the influence of Liuwei Dihuang pills on proliferation and differentiation of rat preadipocytes.
目的探讨SW872前脂肪细胞分化过程中脂联素受体基因表达的变化。
Objective to explore genes expression of adiponectin receptors during differentiation of SW872 preadipocytes.
肥胖症形成的核心是前脂肪细胞增殖分化为成熟脂肪细胞和脂肪细胞中脂质的储积。
The proliferation , differentiation of preadipocyte and the adipogenesis of adipocyte are the core of the development of obesity.
表明茶多酚与茶黄素对前脂肪细胞3T3-L1的增殖和分化具有较好的抑制作用。
These results indicated that proliferation and differentiation of 3T3-L1 preadipocytes were effectively inhibited by tea polyphenols and theaflavins.
目的探讨3'-羟基染料木素对3T3-L1前脂肪细胞增殖、分化的影响及其机制。
Objective To explore the effects of 3'-hydroxygenistein on the proliferation and differentiation of 3T3-L1 preadipocytes and to elucidate the related mechanism.
目的研究黄芩素对3 T3- L 1小鼠前脂肪细胞分化及对脂肪酸合成酶活性的影响。
Objective to study the effects of baicalein on differentiation of 3t3-l1 murine preadipocytes into adipocytes and on the activities of fatty acid synthase (FAS).
目的观察柚皮苷对前脂肪细胞3T3-L1增殖和分化的影响，并探讨其影响3T3-L1分化的可能作用机制。
Objective To observe the effect of naringin on proliferation and differentiation of 3T3 -L1 preadipocyte, and to investigate its possible mechanism.
瘦素抵抗、血清高瘦素浓度等病理状态时，瘦素可能影响前脂肪细胞的增殖及进一步分化为脂肪细胞，调节肥胖发生。
Leptin maybe regulate the generation of obesity through acting on the proliferation and differentiation of preadipocyte at the state of leptin resistance and high leptin concentration in serum.
结果大黄素在较低浓度下对3T3-L1小鼠前脂肪细胞的增殖有一定的促进作用，但当浓度升高时，转化为剂量依赖性抑制作用；
Results Emodin promoted proliferation of 3T3-L1 preadipocyte at low concentration and inhibited the proliferation at high concentration in a dose-related manner.
目的：观察钙离子通道阻滞剂盐酸维拉帕米对3T3鄄L1前体脂肪细胞分化及脂质积聚的影响。
Objective:To observe the effect of verapamil on the differentiation and the lipid accumulation of 3T3-L1 preadipocyte.
结论：在3t3- L1脂肪前体细胞分化过程中脂联素基因表达逐渐上调，可能有利于脂肪细胞的分化成熟。
Conclusions: Adiponectin gene mRNA expression is up-regulated during 3t3-l1 preadipocyte differentiation. It may contribute to the adipocyte differentiation and obesity.
鸡蛋是形成水晶脂肪的胆固醇的好来源，也含有对细胞生长有用的物质，因为鸡蛋是一种正在生长的或“活的”形成前状态。
Eggs are a good resource of cholesterol for the formation of crystalline fat; and also contain substances useful to cell growth as the egg is a growing or "alive" pre-formation state of being.
脂肪组织生长包括脂肪细胞大小的增加和由前体细胞分化的新脂肪细胞的形成。
The growth of adipose tissue includes an increase in adipocyte size and the formation of new adipocytes from precursor cells.
白细胞介素18 (IL - 18)是一种前炎性因子，脂肪组织为其重要来源之一。
Interleukin-18 (IL-18) is a pro-inflammatory factor, and adipose tissue is one of its important sources.
脂肪细胞来源于多能干细胞，其分化经历了多能干细胞、脂肪母细胞、前脂细胞、不成熟脂肪细胞、成熟脂肪细胞几个阶段。
Adipocyte derives from multipotential stem cell. During the course of differentiation, it covers multipotential stem cell, adipoblast, preadipocyte, immature adipose cell, adipocyte.
内源性大麻素物质来源于细胞膜上的脂类前体物质，具有脂类物质的特征，包括氨基化合物和长链脂肪酸。
Endogenous cannabinoids (endocannabinoids), which are synthesized from lipid precursors in plasma membranes, are signaling lipids consisting of amides and esters of long chain fatty acids.
证实胰岛素对原代培养大鼠前体脂肪细胞增殖分化有明显促进作用。
The results demonstrated that the promotion of insulin on proliferation and differentiation of the rat preadipocyte's primary-culture was significant.
脂肪组织来源充足，取材容易而且包含胰岛素前体细胞。
Adipose tissue is abundant and easily accessible and could thus also harbor cells with the potential to differentiate in insulin producing cells.
本文总结了体重减轻、胃肠激素、前肠学说、后肠学说、脂肪细胞因子和炎症因子等方面机制的研究进展。
This review summarizes the possible mechanisms which include weight loss, gastrointestinal hormones, foregut hypothesis, hindgut hypothesis, adipocytokines, and inflammatory factors.
本文总结了体重减轻、胃肠激素、前肠学说、后肠学说、脂肪细胞因子和炎症因子等方面机制的研究进展。
This review summarizes the possible mechanisms which include weight loss, gastrointestinal hormones, foregut hypothesis, hindgut hypothesis, adipocytokines, and inflammatory factors.
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