结论乌司他丁具有一定的抗手术侵袭作用。
Conclusion UTI plays a definite role in resisting operative invasion.
结论乌司他丁对急性轻症胰腺炎有较好的疗效。
Conclusions Ulinastatin has a better effect on mild acute pancreatitis.
目的探讨乌司他丁对创伤性急性肺损伤的作用。
Objective To observe the therapeutic value in traumatic acute lung injury using ulinastatin.
结论：乌司他丁能够有效的辅助治疗失血性休克。
Conclusion: Ulinastatin is effective for the assistant treatment of hemorrhagic shock.
目的探讨乌司他丁对严重烧伤患者脏器功能的作用。
Objective To investigate the protective effects of ulinastatin on organ function in severe burn.
结论乌司他丁可显著改善急性坏死性胰腺炎的预后。
Conclusion Ulinastatin could remarkably improve the prognosis of acute necrotizing pancreatitis.
目的探讨乌司他丁对COPD合并脓毒症患者的治疗价值。
Objective To study the therapeutic value of ulinastatin for patients with COPD complicated with sepsis.
应用抑肽酶和乌司他丁，更好地做好血液保护和心肌保护。
Ulinastatin and aprotinin were good for myocardial protection and blood protection.
本文就乌司他丁在脓毒症时的脏器保护作用的研究作一综述。
This review emphasizes the beneficial effect of ulinastatin on multiple organs in sepsis.
目的探讨乌司他丁(uti)对急性重症胰腺炎的临床疗效。
Objective To investigate the ulinastatin (UTI) on the clinical effect of severe acute pancreatitis.
结论乌司他丁可有效提高HIFU治疗肝癌患者的细胞免疫功能。
Conclusion Ulinastatin can effectively enhance the immunity function of the patients undergoing HIFU treatment for liver cancer.
目的：评价川芎嗪和乌司他丁对断肢再植缺血再灌注损伤的保护作用。
Objective To evaluate the effect of ligustrazine and ulinastatin on protecting ischemia-reperfusion injury after limb replantation.
结论乌司他丁可以减轻CPB引起的脑无氧代谢，改善脑氧摄取与利用。
Conclusion Ulinastatin can alleviate cerebral anaerobic metabolism during CPB by improving the ability of brain tissue oxygen intake and utilization.
目的探讨胰蛋白酶抑制剂乌司他丁对脓毒症大鼠肠黏膜屏障的保护作用。
Objective To investigate the protective role of Ulinastatin UTI on gut barrier of septic rats.
乌司他丁能够保护严重多发伤患者的脏器功能，降低MODS的发生率。
Ulinastati has a protective effect on organ function in Severe Multiple Trauma patients, and can decrease morbidity rate of MODS.
结论乌司他丁对老年人工髋关节置换术患者围术期肝肾功能有明显的保护作用。
Conclusion UTI can protect the liver and kidney function of the aged patient with hip replacement operation.
目的观察乌司他丁(uti)对急性出血坏死性胰腺炎(AHNP)大鼠的治疗作用。
Objective To observe the curative effect of UTI in the treatment of acute hemorrhagic necrotizing pancreatitis (AHNP) rats.
目的探讨区域动脉灌注与静脉输注乌司他丁治疗重症急性胰腺炎（SAP）疗效的差异。
Objective To compare the therapeutic effects of urinary trypsin inhibitor (UTI) administered by regional arterial infusion (RAI) with that by intravenous infusion on severe acute pancreatitis (SAP).
结论乌司他丁对腹部大手术的患者能较好地保护其肝肾功能，减少机体对手术创伤的侵袭。
Conclusion the ulinastatin can protect the hepatorenal function and reduce the operative traumatic reaction for patients with abdominal major operations.
结论乌司他丁可抑制失血性休克大鼠回肠黏膜细胞的凋亡，对失血性休克大鼠起保护作用。
Conclusion Ulinastatin has protective effect on rats with hemorrhagic shock by suppressing the apoptosis in ileal mucosa.
结论：乌司他丁通过抑制细胞因子的生成、减少氧化损伤而对失血性休克大鼠起保护作用。
CONCLUSION: Ulinastatin has a protection effect on rats with hemorrhagic shock by suppressing the production of inflammatory factors and reducing oxidative damage.
方法：40例择期进行CABG的患者随机分为两组：乌司他丁组（A组）和对照组（B组）。
Methods:40 patients undergoing elective CABG were randomly divided into two groups:Ulinastatin group (Group A) and control group (Group B).
目的：探讨乌司他丁对新生鼠缺氧缺血性脑病脑重量及脑细胞变性、坏死、凋亡、胶质细胞增生的影响。
Objective: To study the effect of Ulinastatin on brain weight and cerebrocellular degeneration, necrosis, apoptosis and gliocyte hyperplasia of hypoxic-ischemia encephalopathy in newborn rats.
目的评价乌司他丁对重症脓毒症患者的疗效和安全性，并通过观察患者细胞因子的变化来探讨其作用机制。
ObjectiveTo evaluate the efficacy and safety of ulinastatin in patients with severe sepsis and investigate its mechanisms of action.
结论乌司他丁作为一种新型广谱蛋白酶抑制剂对于治疗急性重症胰腺炎安全、有效、价格低廉，值得临床推广应用。
Conclusion Ulinastatin as a new broad-spectrum protease inhibitor for the treatment of severe acute pancreatitis is safe and effective, low cost, is worthy of clinical application.
结论乌司他丁具有防治缺血性脑水肿、保护脑细胞的作用，作用机制可能与其抑制NOS活性，减少NO的生成有关。
Conclusion Ulinastatin can prevent, treat cerebral edema and protect the cerebral cells by inhibiting NOS activity and reducing NO.
结论乌司他丁具有防治缺血性脑水肿、保护脑细胞的作用，作用机制可能与其抑制NOS活性，减少NO的生成有关。
Conclusion Ulinastatin can prevent, treat cerebral edema and protect the cerebral cells by inhibiting NOS activity and reducing NO.
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