但目前仍未阐明铜中毒时出现MB的机制。
The pathogenesis of MBs in copper toxicosis is still unresolved.
急性铜中毒对金鱼肝、肾细胞结构的这种损伤在解除毒物12天后未见明显恢复。
The liver and renal ultrastructure damnifications of acute copper poisoning goldfish had not evidently comeback by 12 days within innocuity water.
但铜吸收过多可发生铜中毒，如心律失常，严重时会出现肾功能衰竭、尿毒症、休克等。
But absorbing superabundance copper can lead to copper toxicity, inducing arrhythmia, even renal failure, uraemia, and shock.
铜中毒鹅在30天左右，临床病理损害明显，消化道，肝脏，肾脏和心脏是鹅铜中毒毒性损害的靶器官。
Clinical pathological damage of poisoning geese were markedly on 30 days of experiment. The target organs of copper toxic injury were digestive Tract liver, kidney and heart.
铜中毒是指家畜摄食铜元素过多，或铜在肝脏等组织中大量蓄积，因突然释放进入血液循环所引起的一种重金属中毒性疾病。
Copper poisoning is a kind of metal poisoning disease which caused by animal intake too much copper, or remaining a lot of copper in the liver and quickly into blood circulation.
干扰的因素包括铜，铁缺乏和铅中毒。
Causes include copper or iron deficiency and lead poisoning.
用原子分光光度计检测中毒鱼肝、肾、鳃、肌肉中铜的含量，蓄积量的大小顺序是肝脏＞肾脏＞鳃＞肌肉。
Using atomic spectrophotometer examined copper contents of liver, kidney, muscle and gill in subacute poisoned fish. The sequence of accumulation was liver, kidney , gill, muscle.
重金属是一类重要的环境污染物，铜是其中毒性大、分布广的一种。
Heavy metals are important pollutants and copper is one of them with high toxicity and wide-spreaded.
目的探讨慢性铅中毒幼鼠血铅、锌、铜、铁、钙水平变化。
Objective To investigate the changes of lead, zinc, copper, iron and calcium in blood of chronic poisoned infantal mice.
羰基镍是导致铜基合成甲醇催化剂中毒的物质。
Iron and nickel carbonyl compounds are poisons to copper-based methanol synthesis catalysts.
羰基镍是导致铜基合成甲醇催化剂中毒的物质。
Iron and nickel carbonyl compounds are poisons to copper-based methanol synthesis catalysts.
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