大部分与酒精成瘾问题关联的基因是酒精脱氢酶基因(ALDH)，它参与酒精的新陈代谢作用同时对过量饮酒产生保护。
Most consistently associated with alcoholism are the alcohol dehydrogenase genes (ALDH), which are involved in alcohol metabolism and protection against heavy drinking.
自由基学说认为，人体随着年龄增大所发生的退行性变化是细胞正常代谢过程中所产生的过量自由基副作用的结果。
The free radical theory of aging indicated that cellular degeneration was resulted from the excessive free radical that produced in the process of normal cellular metabolism as growth.
代谢综合征和肝癌：与铁过量有关吗？
Metabolic syndrome and liver cancer: Is excess iron the link?
目的以适碘动物为对照(NI)，复制碘缺乏(LI)与碘过量(HI)大鼠动物模型并观察其碘代谢的变化。
Objective to reproduce animal models of iodine excess (HI) and iodine deficiency (li) compared with iodine adequate group (ni) and to observe their IO dine metabolism.
而且，即使制药公司可以找到一种激活棕色脂肪的安全方法，过量的活动会抛开其他代谢系统，可能会损害你的健康。
And even if drug companies could find a way to activate brown fat safely, that excess activity could throw off other metabolic systems that could damage your health.
结果表明，微生物脱磷过程主要是细菌代谢产物对难溶磷酸盐的解离作用和细菌的过量摄磷作用。
The result of researches show that the main processes of dephosphorization are the uptake of by microorganisms and dissociating function of the apatite by the metabolic products of microbes.
应用过量时，可致代谢性碱中毒。
Application of excessive hours can cause metabolic alkalosis.
研究了具有底物和产物抑制以及代谢过量特征的微生物连续培养过程的数学模型。
Mathematic model was studied in continuous cultivation of microorganisms with overflow metabolism, whose growth was inhibited by substrates and products.
脂肪肝是因各种原因导致过量脂肪在肝细胞内堆积，使肝脏功能受损害的一种临床常见代谢性疾病。
Fatty liver is a metabolic disease, caused by excessive fat storing in liver. This disease leads to hypo function of liver.
非酒精性脂肪肝是一种无过量饮酒史，与遗传-环境-代谢应激相关性疾病，导致肝相关性患病率和病死率升高的主要原因之一。
Nonalcoholic fatty liver is a kind of disease related to heredity, environment, and metabolism instead of alcohol. It is a major factor increasing the morbidity or mortality of liver-related disease.
结论过量氧耗除用于血乳酸的消耗外，可能生要用于丙氨酸代谢中的糖新生。
Conclusion we conclude that oxygen consumption after exhaustive maximal exercise is related to glucose resyntbesis through alanine metabolism, as compared with that from lactate metabolism.
结论过量氧耗除用于血乳酸的消耗外，可能生要用于丙氨酸代谢中的糖新生。
Conclusion we conclude that oxygen consumption after exhaustive maximal exercise is related to glucose resyntbesis through alanine metabolism, as compared with that from lactate metabolism.
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