凋亡细胞脾胃湿热组和脾气虚组均高于正常对照组。
The apoptotic cells in both the SSDH and SQD groups were higher than those in the control group.
高级神经中枢参与脾气虚合并胃溃疡疾病过程的调节。
Advanced neural center participates in the regulation during spleen-deficiency combined with gastric ulcer.
目的：探讨脾气虚证、脾阳虚证、脾阴虚证的动物造模及评价方法。
Objective: To explore the animal model-making and evaluation method on syndromes of deficiency of spleen-QI, deficiency of spleen-YANG and deficiency of spleen-YIN.
结论脾气虚证发生有免疫相关基因组学基础，脾虚时机体免疫功能紊乱。
ConclusionThe genesis of Pi-Qi deficiency syndrome has its immune associated genomic basis, and the immune functions are disordered in patients with that syndrome.
目的建立原发性肝癌脾气虚证量化标准（包括量化诊断标准和程度分级标准）。
Objective To set a quantified standard (including quantified diagnosis standard and classification standard) on spleen-deficiency syndrome of primary liver cancer(PLC).
目的：探讨补中益气汤不同浓度、不同时间段对脾气虚证大鼠血清胃泌素的影响。
Objective: To explore the effect of Buzhong Yiqi Decoction (BZYQD) with various concentrations at different time points on serum gastrin in spleen-qi deficiency rats.
同时脾气虚弱，运化失常，产生水湿痰饮等病理产物，成为中风和中风后痴呆的发病根源。
At the same time, temper weak, transport of disorders, resulting in a product of wet phlegm, such as pathology, becoming after a stroke and stroke incidence of the root causes of dementia.
研究慢性胃炎脾气虚证的临床症状、体征和胃镜下胃黏膜象，以探求其宏观及微观诊断要点。
This paper studied the conditions of clinical symptoms, signs and gastroscopic mucosa to explore the diagnostic essentials of the spleen-qi deficiency of chronic gastritis.
建立了小儿过敏性鼻炎脾气虚证辨证预测体征诊断模型，为临床诊断提供了新的研究途径和方法。
So the establishment of indicator syndrome diagnostic model on paediatric Allergic Rhinitis provide new methods and means for clinical diagnosis.
以30例慢性胃病脾气虚患者为实验组，观察其外周血T淋巴细胞亚群的改变，并以60例健康人为对照。
The changes of T lymphocyte subpopulation of peripheral blood were observed in 30 cases of chronic gastropathy with spleen deficiency(Experimental Group, G 1), and in 60 normal cases as control(G2).
肝脏中葡萄糖激酶基因表达情况：番泻叶泻下脾气虚组大鼠肝脏中葡萄糖激酶鑫因表达与生理盐水对照组比较显著降低（P<0 .01）。
Compared with the Saline control groups, the gene expression of glucokinase in the liver on model rats of the senna leaf laxation group remarkably decrease (P<0. 01).
方法：本实验以脾虚sd大鼠模型为载体，选择反应脾气虚主要病理改变的几个生化指标，从一个角度初步探讨该制剂对应力性溢尿症病机的影响。
Methods: spleen deficiency SD rats model is used as carrier in the experiments. Changing several biochemistry's markers that can response primary pathologic alteration of spleen deficiency.
方法：本实验以脾虚sd大鼠模型为载体，选择反应脾气虚主要病理改变的几个生化指标，从一个角度初步探讨该制剂对应力性溢尿症病机的影响。
Methods: spleen deficiency SD rats model is used as carrier in the experiments. Changing several biochemistry's markers that can response primary pathologic alteration of spleen deficiency.
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