小脑性共济失调患者可出现轮替运动障碍。
Individuals with cerebellar ataxia could display dysdiadochokinesia.
该组患者常见恶心，呕吐，头晕和共济失调等症状。
Symptoms such as nausea, vomiting, dizziness and dystaxia were common in this group of patients.
这些症状之后就是共济失调。
结果病毒感染是急性共济失调的主要病因。
Results Virus infection is the main cause resulted in acute ataxia in infant.
最主要的临床表现为眩晕、小脑性共济失调。
The typical clinical manifestations were vertigo and cerebellar ataxia.
共济失调的四种类型。
行走不稳，言语不清，共济失调，肌张力改变。
Their clinical symptom includes walking unstable, speech disorder, ataxia, dystonia.
目的探讨针刺疗法治疗共济失调型脑瘫的疗效。
ObjectiveTo investigate the efficacy of acupuncture in ataxia children with cerebral palsy.
目的总结小儿急性共济失调的常见类型与病因。
Objective To summarize the common types and medical reasons for acute infant ataxia.
症状包括虚弱，共济失调和始发于后肢脚趾的行动不便。
Symptoms such as weakness, ataxia, and dragging of the toes start in the rear legs.
低血压可能发生，作为与冷漠和共济失调症状过量的迹象。
Hypotension may occur as a symptom of overdose with signs of apathy and ataxia.
方法将40例共济失调型脑瘫患者分为观察组和对照组。
Methods40 patients were divided into 2 groups, treatment group and control group.
目的：探索线粒体dna点突变与遗传性共济失调的关系。
Objective: to study the possible relationship between mitochondrial DNA point mutations and hereditary ataxia.
腓肠神经的幅度与疾病持续时间、共济失调评分呈负相关。
Sural amplitude was negatively correlated with disease duration and ataxia score.
目的探讨共济失调毛细血管扩张症的细胞遗传学异常特点。
Objective to investigate the characteristics of the cytogenetic anomalies of ataxia telangiectasia.
其他可能的症状还有共济失调、眩晕、癫痫发作和吞咽困难。
Other possible symptoms are ataxia, vertigo, seizures, and dysphagia.
方法对36例急性共济失调患儿的临床资料进行回顾性分析。
Methods The clinical datas of 36 infant patients suffering from acute ataxia are analyzed retrospectively.
目的探讨感觉性共济失调型CIDP的临床特点和发病机理。
Objective To explore the clinical characteristics and pathogenic mechanism of sensory ataxia form of GBS.
目的研究遗传性脊髓小脑性共济失调7型（SCA7）的基因突变和临床特征。
Objective To study the gene mutation and clinical characteristic of hereditary spinocerebellar ataxia type 7 (SCA7).
临床分期分三期，第一期包含有小脑症状（如共济失调），情感冷漠和坐立不安。
Patients often present in one of three clinical stages. The first stage consists of cerebellar signs (such as ataxia), apathy and motor restlessness.
目的研究细胞凋亡在脊髓小脑性共济失调3型(SCA3)分子发病机制中的作用。
Objective to study the role of cell apoptosis in the molecular pathogenesis of spinocerebellar ataxia type 3 (SCA3).
入院时，患者定向力正常，但表现出严重的构音障碍，左侧中枢性面瘫，共济失调步态。
On admission, he was oriented, but had severe dysarthria, left-sided central facial palsy and gait ataxia.
回顾小脑性共济失调的研究历史、分类，介绍脊髓小脑共济失调的病因机制和治疗方法。
The history of the research of cerebellar ataxia, and the different classifications of ataxia were reviewed. The pathogenesis and treatments of spinocerebellar ataxia were summarizd.
结论纯运动性偏瘫患者，如果出现构音障碍、头晕及共济失调时应考虑到桥脑梗死的可能。
Conclusion The patients with pure motor hemiparesis and dizziness, ataxia, dysarthria have more possibility to suffer from pontine infarction.
急性酒精中毒对中枢神经系统的影响，症状有共济失调、语言含糊、平衡失调、昏迷及呼吸系统衰竭。
Acute alcohol poisoning mostly involves in central nervous system, toxicities including ataxia, slurred speech, loss of equilibrium, coma and respiratory failure.
MS的症状包括视力模糊、平衡缺失、共济失调、极度疲劳、瘫痪和失明。目前尚无可以治愈的方法。
Ms symptoms may include blurred vision, loss of balance, poor coordination, extreme fatigue, paralysis and blindness. There is no cure.
目的总结小儿急性共济失调的常见类型与病因方法对36例急性共济失调患儿的临床资料进行回顾性分析。
Objective To summarize the common types and medical reasons for acute infant ataxia. Methods The clinical datas of36infant patients suffering from acute ataxia are analyzed retrospectively.
更详细的临床检查还发现共济失调和诊断成像，包括前，后的对比计算机断层扫描，发现一个老骨折C2 。
More detailed clinical examination also revealed ataxia, and diagnostic imaging, including pre- and post-contrast computed tomography, revealed an old fracture of C2.
方法应用PCR方法和琼脂糖凝胶电泳对92例不明原因共济失调先证患者测定其SCA1、SCA2和SCA3基因片段长度。
Methods The length of genetic segment of SCA1, SCA2 and SCA3 in 92 patients with ataxia of unknown origin were detected by polymerase chain reaction (PCR) and agarose gel electrophoresis.
方法应用PCR方法和琼脂糖凝胶电泳对92例不明原因共济失调先证患者测定其SCA1、SCA2和SCA3基因片段长度。
Methods The length of genetic segment of SCA1, SCA2 and SCA3 in 92 patients with ataxia of unknown origin were detected by polymerase chain reaction (PCR) and agarose gel electrophoresis.
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