结论：急性光气中毒主要引起急性肺损伤，通过以糖皮质激素为主的综合治疗，可以在短时间内痊愈。
Conclusion: acute phosgene poisoning cause acute lung injury principally, it can be healed in short time by combination treatment mainly included glucocorticosteroid.
方法对19例急性光气中毒患者的临床症状、体征、动脉血气变化和肺影像学改变等临床资料进行综合分析。
Methods To aggregate analyse the clinical symptoms, signs, the changes of arterial blood gas and lung imaging and so on of 19 acute phosgene poisoning patients.
结论：光气中毒后肺mpo活性显著增加，提示肺部有明显的PMN聚集，耗竭pmn后可以缓解光气中毒后肺损伤的程度。
CONCLUSION: The MPO vigor, following phosgene exposure, increases greatly in lungs, indicating an obvious PMN aggregation. Depletion of PMN can relieve acute lung injury induced by phosgene.
整体动物全呼吸道中毒的血液动力学改变与上呼吸道单独受双光气刺激所引起的血液动力学改变循相似规律。
While the intact rabbits were poisoned, they had the similar course of hemodynamic changes as that of those animals with upper respiratory tract irritated alone.
用扫描电镜和透射电镜观察低剂量双光气吸入中毒所致家兔呼吸系统损伤的超微结构改变。
Scanning electron microscope and transmissional electron microscope were used to observe the ultrastructural changes of respiratory tract in rabbits inhaling low concentration of diphosgene.
用扫描电镜和透射电镜观察低剂量双光气吸入中毒所致家兔呼吸系统损伤的超微结构改变。
Scanning electron microscope and transmissional electron microscope were used to observe the ultrastructural changes of respiratory tract in rabbits inhaling low concentration of diphosgene.
应用推荐