The effects of LDL on VSMC proliferation were concentration and time dependent.
在一定浓度和时间范围内，LDL促细胞增殖作用显示量-效与时-效依赖关系。
Conclusion: HPC protective phenomenon occurs in isolated rabbit VSMC during A-R.
结论：离体血管平滑肌细胞存在着预处理保护现象。
TMP; Adhesive molecules; Signal transduction; Vascular smooth muscle cells (VSMC); Rat.
川芎嗪注射液；黏附分子；信号转导；血管平滑肌细胞。
CONCLUSION: Lig shows a pronounced inhibitory effect on VSMC proliferation after balloon injury.
结论：川芎嗪对家兔主动脉球囊损伤平滑肌细胞增殖有显著抑制。
Results VSMC was grown well and fast, the purity is more than 98% which can meet various cell tests.
结果用该法培养细胞传代生长快，细胞纯度达98%以上，足以满足各种细胞试验需求。
Hyaline degeneration and thickening arterial wall with profound by increases in VSMC in renal arteries.
高血压组可见肾入球动脉玻璃样变，小动脉发生了管壁增厚、细胞数目增多；
P27Kipl restrains the proliferation of VSMC obviously and is prospective to become a new means to cure restenosis after PTCA.
P 27蛋白的高表达可显著抑制血管平滑肌细胞增殖，有望成为治疗PT CA后再狭窄的新手段。
AIM: To observe the relationship between aortic atherosclerosis plaque formation and telomerase activation in vascular smooth muscle cells (VSMC).
目的：探讨动脉粥样硬化形成与血管平滑肌细胞端粒酶活性表达的相关性。
Fibrinogen had no obvious effect on VSMC migration in scrape assay and on VSMC migration at random direction in Time-lapse microcinematography (P>0.05).
刮伤实验中细胞迁移速度及慢速显微摄像单细胞随机迁移速度在纤维蛋白原组及对照组细胞均无明显差别（P>0.05）。
Objective to study the effect of atorvastatin on the proliferation of vascular smooth muscle cells (VSMC) and the expression of heme oxygenase-1 (HO-1).
目的探讨阿托伐他汀对血管平滑肌细胞增殖的影响及对血红素氧化酶1 (HO - 1)表达的作用。
Objective: to discuss the mechanism of puerarin possesses the function of proliferation artery smooth muscle cell by observing it's promoting VSMC apoptosis.
目的：观察葛根素促进人脐动脉血管平滑肌细胞凋亡的作用，探讨葛根素抑制平滑肌细胞增殖的机制。
A variety of extracellular stimulating factors plays a significant role on the migration of VSMC, such as vasoactive substances, growth factors and extracellular matrix.
多种细胞因子对血管平滑肌细胞的迁移有明显作用，如血管活性物质、生长因子和细胞外基质等。
In vitro culture of human VSMC as a model of vascular research has played a critical role and continue to provide information in the pharmacology and therapy of vascular diseases.
体外培养的脐静脉内皮细胞做为研究血管的模型对研究药理学和血管疾病起着重要作用并将进一步提供信息。
Conclusion Our results indicated that ghrelin can significantly attenuate the calcification in aorta and VSMC partly through the antagonism with endothelin system in circulation and vascular tissues.
结果提示，生长素可能部分通过拮抗血浆和血管组织局部的内皮素系统效应而产生抑制血管组织和血管平滑肌细胞钙化的作用。
Conclusion Our results indicated that ghrelin can significantly attenuate the calcification in aorta and VSMC partly through the antagonism with endothelin system in circulation and vascular tissues.
结果提示，生长素可能部分通过拮抗血浆和血管组织局部的内皮素系统效应而产生抑制血管组织和血管平滑肌细胞钙化的作用。
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