• Vascular endothelial cell excretes , strong vasoconstrictor.

    血管内皮细胞分泌的强缩血管物质。

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  • It is most commonly used as a nasal vasoconstrictor and an appetite depressant.

    最常被用作鼻血管收缩剂和食欲抑制剂。

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  • CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.

    结论:大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂,细胞信号转导通路MAPK的激活参与其收缩效应。

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  • Endothelin (ET) is a recently discovered potent vasoconstrictor peptide exerting effects through combination with its receptor.

    内皮素(et)是一种血管收缩肽,与et受体结合而发挥作用。

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  • In acutely sympathetically denervated areas, the skin or extremity is warm because of loss of sympathetic vasoconstrictor tone.

    急性失交感神经支配的皮肤或者肢端由于失去了交感性缩血管神经紧张作用,皮肤是温暖的。

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  • Endothelin-1(ET-1) is a powerful intrinic vasoconstrictor, which plays an important role in the development of pulmonary hypertension.

    内皮素(ET)-1是强有力的内源性血管收缩剂,在肺动脉高压的发病机制中发挥重要作用。

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  • Conclusion: Vasoconstrictor can increase tumor blood flow, increase flow velocity, decrease ri and it can be used in tumor hemodynamic study.

    结论:血管收缩剂引起肿瘤血供增加,血液流速增快,阻力指数降低,可用于肿瘤的血流动力学研究。

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  • ACE inhibitors work by inhibiting the conversion of angiotensin I to the potent vasoconstrictor, angiotensin II, thereby improving blood flow and blood pressure.

    ACE抑制剂的工作,通过抑制血管紧张素I的转化可以强化血管,血管紧张素Ⅱ可改善血流和血压。

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  • Conclusion CRA seems to be relate to the renal and splenic hemodynamics, also relate to the histology, the Doppler parameters of portal hypertension and the activity of vasoconstrictor systems.

    结论视网膜中动脉可能与肾脏、脾脏的血流动力学变化有关,同样与组织学、门脉高压多普勒参数和血管收缩系统的活性有关。

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  • The results show; (1) The increased hepatic vasoconstrictor reactivity and the adhesive function of platelets in viral infection are the basic mechanism of the hepatic microcirculatory disturbance.

    结果提示:(1)在病毒感染中肝血管收缩反应性上升及血小板粘附功能增强是肝微循环障碍发生的基础。

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  • The results show; (1) The increased hepatic vasoconstrictor reactivity and the adhesive function of platelets in viral infection are the basic mechanism of the hepatic microcirculatory disturbance.

    结果提示:(1)在病毒感染中肝血管收缩反应性上升及血小板粘附功能增强是肝微循环障碍发生的基础。

    youdao

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