AIM: To reveal ionic current basis of the change of transmembrane potential induced by oxygen free radical.
摘要目的：揭示氧自由基导致跨膜电位变化的离子电流基础。
Objective to investigate the role of reactive oxygen species (ROS) and ROS-caused mitochondrial transmembrane potential loss in sodium selenite-induced apoptosis in NB4 cells.
目的探讨亚硒酸钠诱导NB 4细胞凋亡过程中，活性氧引起的线粒体膜电位丧失及凋亡作用的相关机制。
BACKGROUND Increasing the activity of defective cystic fibrosis transmembrane conductance regulator (CFTR) protein is a potential treatment for cystic fibrosis.
背景提高有缺陷的囊性纤维化跨膜电导调节(CFTR)蛋白的活性是囊性纤维化的一种潜在治疗。
BACKGROUND Increasing the activity of defective cystic fibrosis transmembrane conductance regulator (CFTR) protein is a potential treatment for cystic fibrosis.
背景提高有缺陷的囊性纤维化跨膜电导调节(CFTR)蛋白的活性是囊性纤维化的一种潜在治疗。
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