The expression level of SREBP-1 and leptin increased in HF groups.
模型组肝组织固醇调节元件结合蛋白-1、瘦素表达增加。
With the study of SREBP, people's knowledge on cholesterol homeostasis regulation will promote the developing of cholesterol-controlling drugs.
随着对SREBP领域研究的深入，人们对胆固醇平衡调节将认识更多，并有可能开发出更为有效的控制胆固醇的药物。
We are able to turn off the SREBP gene, which controls the production of fat in the liver, and when this happens the fatty liver goes away but the animal remains obese and still remains diabetic.
我们可以关闭SREBP基因，它可控制肝脏脂肪的生产，当发生这种情况时，脂肪肝可消失，但该动物仍然有肥胖和糖尿病。
We are able to turn off the SREBP gene, which controls the production of fat in the liver, and when this happens the fatty liver goes away but the animal remains obese and still remains diabetic.
我们可以关闭SREBP基因，它可控制肝脏脂肪的生产，当发生这种情况时，脂肪肝可消失，但该动物仍然有肥胖和糖尿病。
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