Rather, miR-17~92 acts to help cells that lack the tumor-suppressing Rb protein to proliferate.
相反，miR-17~92是通过帮助缺乏肿瘤抑制Rb蛋白的细胞增殖而发挥作用的。
Conclusion: the phosphorylation of RB protein can be induced by hyperoxia. Excessive proliferation of LF leads the pulmonary interstitial fibrosis.
结论：高氧可能导致RB蛋白磷酸化(RB灭活)，LF过度增殖最终导致肺间质纤维化。
Monoclonal antibody is produced by immunizing animals with a synthetic peptide corresponding to residues surrounding Ser807/811 of human Rb protein.
单克隆抗体由合成肽段免疫动物产生，该肽段与人Rb蛋白的807/811位丝氨酸邻近残基序列一致。
The tumor is associated with mutations of a protein called Rb, or retinoblastoma protein.
这种肿瘤与一种称作rb的蛋白，或视网膜母细胞瘤蛋白(retinoblastomaprotein, prb)的突变有关。
The tumor is associated with mutations of a protein called Rb, or retinoblastoma protein.
这种肿瘤与一种称作rb的蛋白，或视网膜母细胞瘤蛋白(retinoblastomaprotein, prb)的突变有关。
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