Free radicals, linked to rapid aging, are highly reactive with other molecules, including vital DNA and proteins, the destruction of which can damage or kill cells.
与迅速衰老相关联的自由基很容易与包括维持生命所必需的DNA和蛋白质发生反应，而破坏这些DNA和蛋白质，就可能会损伤或杀死细胞。
Light hitting the proteins produces a pair of free radicals, highly reactive molecules with unpaired electrons.
光击中这种蛋白质产生一对游离的活性基，这是一种非常活跃的有着不成对的电子的分子。
These rogue oxygen atoms can remain on their own or combine with hydrogen atoms to form reactive oxygen species (ROS), which wreak havoc with enzymes and proteins and adversely affect cell function.
这些游离的氧原子能维持它们自己的形式或者和氢原子联合成活性氧(ROS)，ROS能被酶和蛋白分解可逆性影响细胞功能。
Oxidant injury, or oxidative stress, occurs when highly reactive molecules called free radicals attack and damage cellular proteins, lipids (fats) and DNA.
氧化损伤，或氧化应激，出现在被称为自由基的高活性分子攻击和破坏细胞蛋白质，脂类(脂肪)和脱氧核糖核酸时。
Although unmetabolized PAHs can have toxic effects, the major conCErn in animals is the ability of reactive metabolites to bind to proteins and DNA.
虽然非代谢变化的多环芳烃有毒性反应，但主要涉及在动物的蛋白质和DNA的代谢反应的能力。
The results show that the reactive loops of protein inhibitors possess certain features which make the proteins very good inhibitors for corresponding proteases.
研究结果表明，蛋白质抑制剂反应部位所处的环区具有独特的结构，使其能够对相应蛋白酶发挥很高的抑制作用。
We all know about the negatives – reactive oxygen kills cells and essentially damages proteins.
我们都知道负活性氧杀死细胞并从根本上破坏蛋白。
Our reactive fluorescent and luminescent probes, biotins and tag enzymes are used for labeling small drug molecules and biopolymers, e. g, proteins, nucleic acids and carbohydrates;
我们提供反应荧光探针和发光探针，生物素和端粒酶能够应用于标记药物小分子和生物聚合物，如蛋白、核酸以及其他碳水化合物；
Our reactive fluorescent and luminescent probes, biotins and tag enzymes are used for labeling small drug molecules and biopolymers, e. g, proteins, nucleic acids and carbohydrates;
我们提供反应荧光探针和发光探针，生物素和端粒酶能够应用于标记药物小分子和生物聚合物，如蛋白、核酸以及其他碳水化合物；
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