Biotechnology companies spend years figuring out the right combination of mutant host organism and reactor conditions to fashion a protein therapy that is safe and effective.
生物科技公司耗费数年时间，试著找出适当的突变宿主生物与反应环境组合，以得到安全有效的蛋白质治疗方法。
Therefore, inhibiting polyglutamine protein aggregation or deposition would be an important target for HD therapy.
多聚谷氨酰氨的聚集、沉淀可能是该病发病的原因和关键。
This fall, the startup company Tivorsan Pharmaceuticals licensed rights from Brown to the key protein, biglycan, hoping to bring the potential therapy through clinical trials.
今年秋天，新兴的生物制药公司Tivorsan已经从布朗大学得到了这种关键蛋白biglycan的生产许可权，希望能将这种潜在的新疗法带入临床试验阶段。
A novel drug therapy - that mimics the suppression of an antioxidant-promoting protein - kills pancreatic cancer cells, new research reveals.
一种新的研究揭示了，有一种新的药物治疗——模拟抗氧化剂的抑制作用——促进蛋白——能够杀死胰腺癌细胞。
CONCLUSIONS Local mild hypothermia therapy can decrease the levels of serum S-100B protein in patients with cerebral hemorrhage indirectly.
局部亚低温治疗能间接降低脑出血病人血清s- 100 B蛋白水平，从而发挥其脑保护作用。
To study the expression of PTEN, EGFR and P16 protein will build up target gene therapy foundation of astrocytoma.
通过对PTEN、EGFR、P 16蛋白的表达研究，为肿瘤的靶基因治疗策略提供了理论依据。
Objective To study the effects of insulin intensive therapy on the level of C-reactive protein (CRP) and neurological function of the diabetes patients with acute cerebral infarction.
目的研究胰岛素强化治疗对糖尿病并急性脑梗塞患者C反应蛋白水平及其神经功能的影响。
Aim: To investigate the effects of insulin intensive therapy on the level of C-reactive protein (CRP) and outcome of the diabetes patients with acute myocardial infarction (AMI).
目的：探讨胰岛素强化治疗对糖尿病合并急性心肌梗死(ami)C反应蛋白(CRP)水平及预后的影响。
Recent progress in gene engineering and related fields such as gene diagnosis, therapy, bioreactor, protein engineering, metabolism engineering, human genome project, bioinformatics is reviewed.
介绍了基因工程及其在基因检测与基因治疗、生物反应器工程、蛋白质工程与代谢工程，基因组计划与生物信息学等相关领域的进展。
That is probably also why protein C supplementation is such a promising potential therapy for septic shock.
这也是补充蛋白C是治疗内毒素休克一项极具希望疗法的所在。
To compare the effects between gene therapy and gradual release carrier for bone morphogenetic protein-2 (BMP-2) in repairing bone defects.
比较骨形态发生蛋白2 (BMP - 2)基因治疗与生长因子缓释方法修复节段性骨缺损效果。
Polysaccharide Krestin (PSK), a kind of protein-bound polysaccharide extracted from Coriolus Versicolor, is used as a immune modulator in anti-tumor therapy.
云芝多糖是一种从云芝子实体提取的蛋白结合多糖，已被广泛用作肿瘤治疗的免疫佐剂。
Polysaccharide Krestin (PSK), a kind of protein-bound polysaccharide extracted from Coriolus Versicolor, is used as an immune modulator in anti-tumor therapy.
云芝多糖是一种从云芝子实体提取的蛋白结合多糖，已被广泛用作肿瘤治疗的免疫佐剂。
We got satisfactory effect by using non - drug therapies such as physical stimulation, allotransplantation of protein or injecting minute dosage of drug into acupoints, dialysis, heat therapy.
应用物理刺激、异体蛋白植入或极小剂量药物穴位注射、透析、热疗等非药物方法治疗疾病，取得了满意的效果。
Objective to probe into the mechanisms of bone morphogenetic protein-2 (BMP-2) in osteosarcoma and to provide basis for gene therapy.
目的从分子水平探讨骨形态发生蛋白2 (BMP - 2)基因对骨肉瘤生物学行为的影响，并为骨肉瘤的基因治疗提供理论依据。
Objective To study biochemical function and therapy of the NP (nucleocapsid protein) gene of NDV (new castle disease virus). Methods the purified genome RNA of NDV were used for template.
目的研究新城疫病毒(NDV)核衣壳蛋白(NP)基因的生物学作用及其抗人喉鳞癌的作用。
Genetic treatment, protein replacement therapy and bone marrow transplantation are the main forms of treatment for EB-related injuries.
治疗EB目前主要尝试的方法是基因疗法、蛋白替代疗法和骨髓移植。
As a matter of fact, the compounds of the formula (I) are useful, in therapy, in the treatment of diseases associated with a disregulated protein kinase activity, like cancer.
事实上，式（I）化合物在疗法中可用于治 疗与失调的蛋白激酶活性相关的疾病例如癌症。
Objective To study biochemical function and therapy of the NP (nucleocapsid protein) gene of NDV (new castle disease virus).
目的研究新城疫病毒(NDV)核衣壳蛋白(NP)基因的生物学作用及其抗人喉鳞癌的作用。
Conclusion OD-PTD fusion protein can significantly inhibit the proliferation of K562 cells, which provides an experimental basis for new therapy of chronic myeloid leukemia.
结论OD - PT D融合蛋白对K562细胞有明显的抑制作用，为进一步探讨慢性粒细胞白血病新的治疗手段提供了实验依据。
Conclusion OD-PTD fusion protein can significantly inhibit the proliferation of K562 cells, which provides an experimental basis for new therapy of chronic myeloid leukemia.
结论OD - PT D融合蛋白对K562细胞有明显的抑制作用，为进一步探讨慢性粒细胞白血病新的治疗手段提供了实验依据。
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