Objective To investigate the change of intercalated discs induced by pressure overload.
目的研究压力超负荷时心肌闰盘的变化。
Essential hypertrophy; Left ventricular hypertrophy; Rat with pressure overload; Xinjikang.
高血压；左心室肥厚；压力负荷性大鼠；心肌康。
AIM: To investigate the alterations of cardiac mechanics and cyclic nucleotide in rats after pressure overload.
目的：探讨大鼠压力超负荷后心肌力学及环核苷酸的变化规律。
Conclusion: High dose of Herba Siegesbeckiae extract has a protection effect on myocardial remodeling in rats with pressure overload.
结论：高剂量的豨莶草提取物对压力超负荷型心肌重构具有一定的保护作用。
Objective: To evaluate the function and configuration changes of the ventricle under pressure overload of the contralateral ventricle.
目的：探讨一侧心室压力负荷时，对对侧心室功能及结构的影响。
AIM: To compare the evaluations for the structure and function of the hypertrophic hearts induced by volume overload or pressure overload in rats.
目的：通过建立压力负荷型和容量负荷型心脏肥大大鼠模型，比较各种评价大鼠心脏结构和功能的方法。
Objective To investigate the relationship between pressure overload and morphological remodeling of cardiac, pulmonary and renal arterioles in rats.
目的探讨压力超负荷与大鼠心、肺、肾内小动脉形态学重建的关系。
Conclusion Under chronic pressure overload, the activated ASK1 with the increasing ROS may play an important role in signal transduction pathway of myocardial hypertrophy.
结论压力超负荷下随着ROS的升高，激活的ASK1可能在心脏肥大信号传导途径中发挥重要作用。
Objective: To study on the plasma level of at rial natriuretic polypeptide (ANP) and effect of digoxin in the rats with chronic congestive heart failure (CHF) by pressure overload.
目的：观察后负荷增加所致充血性心力衰竭(CHF)大鼠血浆心钠素(ANP)水平的变化及地高辛的影响。
BACKGROUND: Pressure overload can lead to the activation of renin-angiotensin-aldosterone system, and then left ventricular remodeling occurs. Captopril can reverse this progress.
背景：压力负荷可以导致肾素血管紧张素醛固酮系统激活，进而导致左室重构，卡托普利对这种重构具有逆转作用。
Objective To study the characteristics of heart structure changes in rat model with hypertrophic cardiomyopathy (HCM) induced by pressure-overload, and the interference of berberine.
目的研究压力超负荷致心肌肥厚模型大鼠心脏结构的变化特点及小檗碱对此的干预作用。
To investigate the effect of atorvastatin on gap junction remodeling in hypertrophied myocardium induced by pressure-overload.
目的探讨阿托伐他汀对心肌肥厚时缝隙连接重构的影响。
AIM: To investigate the role of phosphoinositide pathway in the formation of pressure-overload cardiac hypertrophy.
目的：探讨肌醇磷脂途径在压力超负荷性心肌肥厚形成中的作用。
AIM: To investigate the role of phosphoinositide pathway in the formation of pressure-overload cardiac hypertrophy.
目的：探讨肌醇磷脂途径在压力超负荷性心肌肥厚形成中的作用。
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