The cellular distribution of PKC and ankyrin was observed by indirect immunofluorescence.
采用间接免疫荧光标记方法观察PKC及锚蛋白的细胞内分布情况。
In our experiments, levels of PKC and GDNF decreased.
在我们的实验中，PKC和gdnf水平均下降。
PKC activity was measured with phosphoryl abevacuation .
磷基转移法测定PKC活性变化。
Protein kinase C(PKC)activity of cell membrane was detected.
放射性同位素法检测肺癌细胞蛋白激酶C（PKC）活性。
These certificates are used to establish the trust of any inbound PKC.
这些证书用于建立任何入站PKC的信任关系。
The PKC activity was determined by competed protein binding method.
竞争蛋白结合法测定PKC的活性；
PKC Group has two business areas: Wiring Harnesses and Electronics.
比克希集团有线束及电子两大业务单元。
Public Keys are distributed in containers called public key certificates (PKC).
公有密钥分布于名为公钥证书（PKC）的容器中。
PKC activation may be one of the mechanism of AP intensified by hyperlipaemia.
PKC的活化可能是高脂血症加重急性胰腺炎的机制之一。
Expression of AQP4 and PKC in brain tissue was detected by immunohistochemistry.
AQP4和PKC的表达使用免疫组织化学方法检测。
The Port Kembla Copper (PKC) copper smelting facilities were commissioned during 2000.
改建的肯布拉港铜冶炼厂2000年投入运行。
Objective To study platelet protein kinase C(PKC)′s effect on platelet activity and its role.
目的研究急性脑梗死血小板蛋白激酶C（PKC）对血小板活性的影响及其致病作用。
There are some relations between PKC and GDNF, but how they regulate each other, need further research.
在PKC和GDNF之间存在着一定的联系，它们是如何相互调节的，尚需进一步研究。
Truststores — trusted certificate entry: This type of entry contains a single PKC belonging to another party.
信任存储库——受信任的证书条目：这种类型的条目包含单个属于另一方的PKC。
Several aspects need to be considered for the successful clinical development of PKC inhibitors in NSCLC.
PK C抑制剂在NSCLC中成功的临床应用还需要考虑到几个方面。
At present, 12 kinds of PKC isoforms have been dissociated and purified, and they are coded by different genes.
目前已分离、纯化出至少12种不同的PKC亚型，它们是由不同基因编码的功能同功酶。
It has been shown that its protective effect is related to protein kinase C (PKC) and extracellular signal-related kinase (ERK).
研究表明其保护效应与蛋白激酶C（PKC）和胞外信号调节激酶（ERK）有关。
This function is accomplished through activating the PKC, and the expression of CD133 is regulated possibly after transcription.
这一作用主要通过活化蛋白激酶C（PKC）通路来实现，而CD133抗原表达可能是转录后进行调节。
Conclusion Decreased PKC positive cells in the preoptic area may be associated with endocrine disturbance in postmenopausal women.
结论下丘脑视前区PK C阳性细胞的减少可能在绝经后女性内分泌失调中起重要的作用。
Objective To investigate the effects of blocking renin-angiotensin system on protein kinase C (PKC) activity in diabetic rat kidney.
目的观察阻断肾素血管紧张素系统对糖尿病大鼠肾组织蛋白激酶C（PKC）活性的影响。
In recent years, studies have found that there is a certain correlation between the expressions of AQP4 and PKC after ischemic stroke.
近年来的研究发现，缺血性卒中后aqp4与PKC的表达呈现一定的相关性。
This article re - views the distribution, function, functional regulation of AQP4 and PKC, and their relations in ischemia-reperfusion.
文章就AQP4与PKC的分布、功能、功能调节及其在缺血再灌注中的关系进行了综述。
This paper presents the representation of components of the enciphering and deciphering transformation in LUC PKC, and gives its proof.
提出了LUC公钥密码体制加解密变换分量表示，并给予了证明。
Objective Inquiry into the clinical characteristics and treatment respond of patients with paroxysmal kinesigenic choreoathetosis (PKC).
目的研究发作性运动性舞蹈徐动症的临床特征与治疗反应。
It appears that PKC inhibitors act on similar and, in some instances, distinct pathways compared to those of the EGFR and VEGFR inhibitors.
PK C抑制剂似乎以相同的方式发挥作用，在某些情况下与那些EGFR和VEGFR抑制剂比较，却有不同的作用途径。
This review mainly summarized the PKC effects on the cell growth, proliferation, cell cycle and telomerase of nasopharyngeal carcinoma (NPC).
本文就PK C对鼻咽癌细胞生长、增殖、细胞周期及端粒酶等的影响作一综述。
PKC is an important element of cellular signal transduction pathways that regulate cell growth, survival, differentiation and gene expression.
PKC是细胞信号传导途径中的一个中心环节，调节细胞的基因表达、生长发育及分化。
Objective:To explore the effect of tetramethylpyrazine (TMP) on protein kinase C (PKC) expression of pulmonary arteries in chronic hypoxic rats.
目的：观察川芎嗪对慢性缺氧大鼠肺动脉蛋白质激酶C（PKC）表达的影响。
The up-regulation and redistribution of TorsinA is not caused by the pathway of the PMA activating PKC after cells insulted by oxidative stress.
结论扭转蛋白A可能是膜相关蛋白，细胞氧化应激中扭转蛋白A表达上调和重分布变化不是由佛波酯诱导的蛋白激酶C活化途径来实现的。
The up-regulation and redistribution of TorsinA is not caused by the pathway of the PMA activating PKC after cells insulted by oxidative stress.
结论扭转蛋白A可能是膜相关蛋白，细胞氧化应激中扭转蛋白A表达上调和重分布变化不是由佛波酯诱导的蛋白激酶C活化途径来实现的。
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