Anything from cosmic rays to radiation to diet may activate a dormant oncogene.
从宇宙射线到辐射到日常饮食，任何东西都可能激活潜伏的致癌基因。
Aberrant somatic hypermutation and deregulation of the oncogene BCL-6 are associated with lymphomagenesis.
异常的超变和异常的致癌基因 BCL-6 与淋巴瘤的发生具有相关性。
Now the study on oncogene is not limited in oncology, and it is involved in cell survival, proliferation, differentiation and information transfer.
目前有关癌基因的研究已不仅局限于肿瘤学，癌基因对正常细胞的生长、增殖、分化以及细胞内信息传递亦有重要意义。
Contrast with proto-oncogene and with tumor-suppressor gene.
相对于原癌基因和肿瘤抑制基因。
The inactivation of MGMT gene may urge the proto oncogene activation.
MGMT基因的失活常导致原癌基因的激活。
It is the first tyrosine phosphatase that was defined as the oncogene.
是第一个被定义为癌基因的酪氨酸磷酸酶。
The protein expression of oncogene was assayed with immunohistochemistry.
免疫组化检测癌基因蛋白表达。
The stem cell leukemia(SCL) gene is a new oncogene related with leukemogenesis.
干细胞白血病（SCL）基因是新发现的与白血病发生有关的癌基因。
Conclusion Mutation of RET proto oncogene might play an role in the familial Hirschsprung's disease.
结论RET基因突变与家族性先天性巨结肠症的发生有关。
Here we show that oncogene-induced senescence occurs in otherwise normal murine hepatocytes in vivo.
在此我们发现癌基因诱导的衰老在正常小鼠的活体肝细胞中同样存在。
A prefix of "v-" before oncogene symbols indicates oncogenes captured and transmitted by RETROVIRUSES;
癌基因符号前的前缀“v-”表明获得性及由反转录病毒传递的癌基因。
Mutational activation of the BRAF oncogene is the most common genetic alteration in cutaneous melanoma.
BRAF癌基因突变的激活是皮肤黑色素瘤最常见的基因改变。
Anything from cosmic rays to radiation to diet may activate a dormant oncogene, but how remains unknown.
从宇宙射线、辐射到日常饮食，任何东西都可能激活一个处于静止状态下的致癌基因，但如何激活尚不为人知。
Aberrant somatic hypermutation and deregulation of the oncogene BCL-6 is associated with lymphomagenesis.
异常的超变和异常的致癌基因BCL-6与淋巴瘤的发生具有相关性。
Conclusion: The mRNA overexpression of MET proto-oncogene is correlated with tumor differentiation in ESCC.
结论：MET原癌基因表达水平与食管鳞癌细胞分化相关。
VEGF121 expression is induced by hypoxia, inflammatory cytokines and through oncogene products in tumors (1-3).
VEGF121的表达受到组织缺氧，炎症因子和肿瘤组织产生的致癌基因的诱导（1-3）。
Objective To investigate the relationship between RET proto oncogene and the pathogenesis of Hirschsprung′s disease.
目的探讨原癌基因RET与先天性巨结肠发生的关系。
Objective: to investigate the changes in histogenesis and expressions of related oncogene protein in recurrent bone tumor.
目的：探讨骨肿瘤复发后组织起源及相关癌基因蛋白表达的变化。
Pituitary tumor transforming gene 1 (PTTG1) is a proto-oncogene and plays roles in promoting tumorigenesis and metastasis.
垂体肿瘤转化基因1 (PTTG1)具有促进肿瘤生长和转移的作用。
The occurrence mechanisms on cancer including mutation of oncogene and antioncogene, effect of environmental factors were reviewed.
根据近年的研究进展，从癌基因与抑癌基因的突变及环境因素的影响等方面综述了癌症的发生机理。
The influencing of some oncogene and anti-oncogene to the expression and activity of HIF-1 determines neoplastic prognosis ultimately.
而某些癌基因和抑癌基因对HIF-1表达及活性的影响最终决定着肿瘤的预后。
Objective to investigate the relationship between the expression of RET proto oncogene and the pathogenesis of Hirschsprung's disease.
目的探讨RET基因的表达情况与先天性巨结肠发生的关系。
Purpose:Find the relationship between the expressions of P21 oncogene, P16 antioncogene and the biological action of the bladder cancer.
目的：探讨癌基因P2 1和抑癌基因P16的表达与膀胱癌生物学行为之间的关系。
More and more evidence indicated that the formation of tumor is due to multi factors with oncogene activation and anti oncogene inactivation.
越来越多的证据表明，肿瘤的发生是多因素作用的结果，与癌基因的激活与抑癌基因的失活有关。
The EBV latent membrane protein (LMP1) gene and nucleoantigen gene EBNA2 are virus-oncogene, which can induce malignant transformation of cells.
EBV潜伏膜蛋白基因LMP1和核抗原基因EBNA2是公认的导致细胞恶性转化的病毒癌基因。
Programmed cell death factor 4 (PDCD4) gene is a new anti-oncogene, which is firstly identified as a specific protein expressed in cell apoptosis.
程序性细胞死亡因子4 (PDCD4)基因是首先在细胞凋亡中因一种表达特异的蛋白质而鉴别出来的一种新的抑癌基因。
Objective to investigate the effect of antisense nucleotide of human cervical cancer oncogene (HCCR) on the cell proliferation and apoptosis of HepG2 cells.
目的探讨针对人宫颈癌基因(HCCR)反义核苷酸影响肝癌细胞增殖、凋亡的作用。
Quite a few kinds of genes such as susceptibility gene, oncogene and tumor suppressor genes have been found with implications for diagnosis, therapy and prognosis.
其中涉及多种基因异常，包括易感基因、癌基因、抑癌基因等，对于乳腺癌的诊断、治疗及预后判断均具有提示作用。
Expression of an oncogene in a primary cell can, paradoxically, block proliferation by inducing senescence or apoptosis through pathways that remain to be elucidated.
初始细胞的癌基因的表达往往会通过诱导细胞凋亡、衰老来阻止细胞的增值的，而其实现上述功能的具体途径仍有待澄清。
The mechanism of tumor dormancy is related to hindrance of angiogenesis, oncogene and antioncogene, immunocompetence, proliferation cycle, signal conduction and so on.
其发生机制可能与肿瘤血管生成受阻、癌基因与抑癌基因、宿主免疫状态、肿瘤细胞增殖周期及信号传导有关。
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