All this suggested that the NCAM, which regulated by light exposure, might be involved in post hatch learning and memory behaviour.
这一结果提示，不同光照条件在孵化过程中，对NCAM表达的影响，参与了光照对小鸡后天学习记忆行为改变的过程。
Conclusion NCAM can mediate the protective effect of GDNF on DA neurons, and this role may be related to the increase of CB expression.
结论NCAM参与了GDNF保护DA能神经元的作用，该保护作用可能是通过增加CB的表达而完成的。
Objective to investigate the expression of the neural cell adhesion molecule (NCAM) in human astrocytomas and evaluate the clinic signification.
目的研究人脑星形细胞瘤中神经细胞黏附分子的表达情况及其临床意义。
The expressions of PSA-NCAM in cerebral cortex and hippocampus of the PNE offsprings were higher than the control group while the expressions of NCAM-180,140 and 120 weaker.
与对照组相比，妊娠期暴露尼古丁小鼠的后代大脑皮层和海马中，PSA-NCAM表达升高，而NCAM-180、140和120的表达减弱。
It is indicated that NCAM and GDNF may be involved in the changing process of cerebral plasticity after cerebral ischemia injury, also associated with the functional recovery.
提示神经细胞黏附分子和胶质细胞源性神经营养因子可能参与了脑缺血损伤后脑的可塑性变化过程且与功能恢复有关。
MAIN OUTCOME MEASURES: The cellular density and cross section area of RMS, the expression of polysialylated neuronal cell adhesion molecule (PSA-NCAM)RESULTS: Totally 25 SD rats were recruited.
主要观察指标：嗅茎后份平面的细胞密度，横截面积和唾液酸-神经细胞黏附分子的表达。
MAIN OUTCOME MEASURES: The cellular density and cross section area of RMS, the expression of polysialylated neuronal cell adhesion molecule (PSA-NCAM)RESULTS: Totally 25 SD rats were recruited.
主要观察指标：嗅茎后份平面的细胞密度，横截面积和唾液酸-神经细胞黏附分子的表达。
应用推荐