Blood LAA was positively correlated with tissue MPO content.
外周血laa与肺组织M PO含量呈正相关。
The activity of myeloperoxidase(MPO) in liver tissue was also determined.
测定肝组织中髓过氧化物酶（MPO）含量。
Administration of pentoxifylline significantly reduced the blood MPO level.
己酮可可碱可显著降低血清mpo水平。
The secondary lysosomes and the small granules are strongly positive to MPO.
次级溶酶体和小颗粒呈MPO强阳性。
Chlorotyrosine was not elevated in patients with high MPO or carbonyl levels.
M PO或蛋白质羰基升高的病人3 -氯酪氨酸并不升高。
The relation between MPO activities and complicated cardiovascular disease was analyzed.
横断面研究MPO活性与合并心血管疾病之间的相关性。
The myeloperoxidase (MPO) was also assayed to reflect the magnitude of PVEC-PMN adhesion.
测定髓过氧化酶（MPO）活性，反映PVEC粘附PMN的数量。
To study the clinical significance of anti-PR3 and anti-MPO s detection in systemic vasculitis.
探究抗蛋白酶3（PR3）抗体、抗髓过氧化物酶（MPO）抗体在系统性血管炎患者中检测的临床意义。
MAIN OUTCOME MEARSURES: Positive expression of ICAM 1, MPO activity and cellular ultrastructure.
主要观察指标：ICAM -1阳性表达，MPO活性，细胞超微结构。
Conclusion the MPO blood level positively correlates with the cytokine levels and the severity of AP.
结论血清mpo水平与血清细胞因子水平和AP的严重程度呈正相关。
The difference in immunological characteristics of MPO-ANCA might contribute to different disease expression.
抗mpo抗体免疫学特性的差异可能与患者不同临床表型相关。
The NO content in serum and myeloperoxidase (MPO) content in brain tissue were determined to study the its mechanisms.
同时测定大鼠血清NO及脑组织中MPO含量以探讨氧化苦参碱的作用机制。
Measured in the experiment after the end of the colon length, tissue injury score (HS), myeloperoxidase (MPO) activity.
并在实验结束后测量结肠长度、髓过氧化物酶(mpo)活性、组织损伤评分(HS)。
MPO was positive in HL60 cells. K562 cells morphology obviously showed a primitive and no-differentiation characteristic.
细胞形态学上K562细胞原始未分化特性比较明显，HL60细胞质内已有嗜天青颗粒和空泡等细胞分化特征。
Results: Compared with the model group, liquiritin noticeably decreased the activity of MPO and the expression of ICAM-1.
结果：与模型组相比，甘草苷可明显减轻脑组织病理学改变，降低MPO活性及ICAM - 1表达。
Objective To investigate the changes of interleukin 10 (IL 10) and MPO and their relation in acute myocardial ischemia and reperfusion.
目的探讨大鼠心肌缺血及再灌注过程中血清白介素10 (IL 10)、心肌组织中髓过氧化物酶(mpo)活性的变化和相互关系。
Conclusion as sensitive markers of systemic vasculitis, anti-PR3 and anti-MPO antibody may be useful for diagnosis and early treatment.
结论PR 3、MPO抗体作为系统性血管炎的一种敏感标记抗体，有利于该疾病的早期治疗。
The efficacity of MPO significantly grew after hemorrhage ( P<0.05), while in the group of treatment , MPO was suppressed significantly.
MPO活性在失血后显著升高（P＜0.05），而治疗组的活性受到抑制。
Myeloperoxidase (MPO) is found to be associated with many kinds of diseases, such as inflammation, lung cancer, leukemia, and arteriosclerosis.
髓过氧化物酶与很多疾病有关联，其功能和遗传变异可引起很多疾病，如炎症、肺癌、白血病和心血管疾病。
C5 fragment induced MPO and lactoferrin release was produced in an incremental manner with elastase, however a biphasic response with cathepsin G.
随着弹性酶浓度增加，C5片段导致中性粒细胞释放MPO和乳铁传递蛋白增加，而组织蛋白酶G对两者的释放产生双向反应。
The histopathological change, myeloperoxidase(MPO) activity and intercellular adhesion molecule-1(ICAM-1) expression were evaluated by various methods.
采用不同方法评价脑组织病理学，变化、髓过氧化物酶（MPO）活性及细胞间粘附因子-1（ICAM-1）表达。
Objective to study the pathological characteristics of leukocyte myeloperoxidase (MPO) deficiency and clinical laboratory diagnostic strategy for this disease.
目的探讨白细胞髓过氧化物酶(mpo)缺陷症的病变特点及临床实验诊断策略。
Methods Expression of MPO was detected in bone marrow mononuclear cell (BMMNC) from 36 cases with ANLL and 30 normal controls by capillary electrophoresis (ce).
方法采用毛细管电泳(CE)电化学检测法检测36例ANLL患者及30例正常骨髓单个核细胞(BMMNC)中MPO表达。
Results C5 exposed to cathepsin G and elastase generated C5 fragments. C5 fragments stimulated primed neutrophil to release myeloperoxidase(MPO) and lactoferrin.
结果C5暴露于组织蛋白酶G和弹性酶时，产生C5片段，而C5片段能使活化的白细胞释放MPO和乳铁传递蛋白。
Methods the weight of thrombus was measured, the change of no and MPO in serum and venous vessel wall were determined through the model of inferior vena cava ligation.
方法通过小鼠下腔静脉结扎，测定形成血栓的重量及血清和血管壁内NO与MPO水平的变化。
After adjusted by the C-reactive protein level in multivariate model, the elevated MPO activity of 10 units was attributed to an increased cardiovascular risk for 20.4%.
在多变量模型中，经c -反应蛋白水平校正后，MPO活性每增加10单位，合并心血管疾病的危险增加20.4%。
Objective to evaluate the monitoring action of the expression myeloperoxidase (MPO) and superoxide dismutase (SOD) activity in the active inflammatory bowel disease (IBD).
目的探讨髓过氧化物酶(MPO)和超氧化物歧化酶(SOD)作为炎症性肠病(IBD)病情活动临床监测指标的价值。
Objective: to evaluate the monitoring action of the expression myeloperoxidase (MPO) and superoxide dismutase (SOD) activity in the active inflammatory bowel disease (IBD).
目的：研究髓过氧化物酶(MPO)和超氧化物歧化酶(SOD)活性在监测炎症性肠病(IBD)病情活动中的作用。
The contents of malondialdehyde(MDA), the activity of superoxidase dismutase(SOD), and the activity of myeloperoxidase(MPO) in intestinal mucosa were measured respectively.
比较各组大鼠肠黏膜丙二醛（MDA）含量、超氧化物歧化酶（SOD）和髓过氧化物酶（MPO）的活性变化；
The contents of malondialdehyde(MDA), the activity of superoxidase dismutase(SOD), and the activity of myeloperoxidase(MPO) in intestinal mucosa were measured respectively.
比较各组大鼠肠黏膜丙二醛（MDA）含量、超氧化物歧化酶（SOD）和髓过氧化物酶（MPO）的活性变化；
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