One group of scientists has proposed that alpha-mangostin, can cause cell death in leukemia cells.
一组科学家提出，倒捻子素的抗氧化剂可以导致白血病细胞死亡。
MDM2 increased drug resistance of leukemia cells to ADM.
MDM2增加了白血病细胞对阿霉素的抗药性。
In vitro cytotoxicity was based on culturing P388 leukemia cells.
以P 388白血病细胞的体外培养确定细胞毒活性。
To investigate the mechanisms of mitoxantrone killing leukemia cells.
目的探讨米托蒽醌治疗白血病的机制。
The regrowth ability of primary leukemia cells was lower than leukemia cell line.
原代白血病细胞的再增殖能力低于白血病细胞系。
Objective To explore the regulation mechanism of EDAG expression in leukemia cells.
目的探讨胚胎发育相关基因（E DAG）在白血病细胞中的调控机制。
However, the residuary leukemia cells of autogenous graft can cause leukemia relapse.
然而，自体移植物中残留的白血病细胞可引起白血病复发。
Objective To investigate the expression of EDAG-1 in human myeloblast leukemia cells.
目的研究EDAG 1在人髓系白血病细胞株中的表达情况。
Objective: To investigate the effects of curcumin on human acute myelocytic leukemia cells in vitro.
前言：目的：研究姜黄素在体外对原代白血病细胞的抑制作用。
This study was aimed to investigate the effect of D-limonene on K562 leukemia cells and its mechanism.
本研究探讨d -柠檬烯对K562白血病细胞增殖的影响及机制。
Objective: To investigate the induction of differentiation by tadpole extract (T871) on leukemia cells.
目的：探讨蝌蝌提取液(T871)对白血病细胞的诱导分化作用。
The leukemia cells grow and survive better than normal cells and, over time, they crowd out normal cells.
白血病细胞的生存较正常细胞更好，随着时间的推移，它们会逐步取代正常细胞。
These leukemia cells grow and survive better than normal cells and, over time, they crowd out normal cells.
这些白血病细胞生长快，并且较正常的细胞有更强的存活能力，随着时间的推移，癌细胞的生长超过了正常细胞。
A very high number of leukemia cells building up in the marrow also can lead to low neutrophil and platelet counts.
骨髓中大量白血病细胞的生成也会造成中性粒细胞和血小板计数的降低。
OBJECTIVE To study the mechanism of inhibition of K562 leukemia cells proliferation by glycyrrhetinic acid (GA).
目的：探讨甘草次酸(GA)抑制K562细胞增殖的机制。
When you have leukemia, the bone marrow starts to make a lot of abnormal white blood cells, called leukemia cells.
当你患白血病时，骨髓开始制造大量的异常白细胞，称为白血病细胞。
Objective to set up a sensitive triple color flow cytometric assay (FCM) for detection of residual leukemia cells.
目的建立三染色流式细胞术检测微量残留白血病细胞。
Objective: to study the inhibitory effects of allicin and its induction of apoptosis on human leukemia cells HL-60.
目的：探讨大蒜素对人白血病HL - 60细胞生长抑制和诱导凋亡作用。
Part of the acute promyelocytic leukemia cells appeared indigo granules and apoptosis body in plasma through NBT test.
NBT还原实验发现部分早幼粒白血病细胞胞奖内出现紫蓝色颗粒，并见到核碎解成染色质小体（凋亡小体）。
This fusion protein prevents certain blood cells from maturing and leads to an accumulation of immature leukemia cells.
这一融合蛋白阻止特定血液细胞分化成熟，使幼稚细胞堆积。
Risk was also higher for patients with a high count of immature leukemia cells and a low number of platelet cells in the blood.
那些血液中含有高水平的不成熟的白血病细胞及低水平的血小板细胞的患者的风险更大。
Objective To investigate if CYP3A5 gene is involved in the molecular mechanisms for multiple drug resistance in leukemia cells.
目的探讨CYP3A5基因与白血病细胞多药耐药的关系。
The agglutination test showed that PNA agglutinates neuraminidase-treated erythrocytes, thymocytes, and acute lymphatic leukemia cells.
凝集试验表明PNA能凝集NA处理的红细胞、胸腺细胞及急性淋巴细胞性白血病细胞；
A specific immune response was detected in the bone marrow, accompanied by loss of normal B cells and leukemia cells that express CD19.
在骨髓中检测到特异性免疫反应，伴随着表达CD19的正常B细胞和白血病细胞的损失。
Objective To investigate the effects of quercetin on cell morphology and VEGF expression of acute myeloblastic leukemia cells NB4 in vitro.
目的探讨槲皮素对NB 4细胞的形态、血管内皮生长因子(VEGF)表达的影响。
In prior studies, Sipkins found that leukemia cells and some solid tumors create specific niches in the bone marrow where they multiply and spread.
在早先的研究中，斯普金思发现白血病细胞和其它一些固态肿瘤细胞在骨髓里创建特殊的栖身处所，并在那里增殖和传播。
Objective to investigate the gene transduction efficiency of lentiviral vector in leukemia cells to provide key basis for leukemia gene therapy.
目的：探讨慢病毒载体在白血病细胞中的基因转导效率，为白血病基因治疗提供关键依据。
Conclusion 12g5 can change the biological properties of HL-60 cells and inhibit proliferation and survival of leukemia cells at a certain level.
结论12g5可改变HL 60细胞的生物学特性，从而在一定程度上抑制白血病细胞的增殖，影响细胞生存。
Conclusion 12g5 can change the biological properties of HL-60 cells and inhibit proliferation and survival of leukemia cells at a certain level.
结论12g5可改变HL 60细胞的生物学特性，从而在一定程度上抑制白血病细胞的增殖，影响细胞生存。
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