Objective To study the effects of ATP sensitive potassium channel (KATP) opener on focal cerebral ischemia-reperfusion.
目的探讨AT P敏感性钾通道开放剂对大鼠局灶性脑缺血再灌注损伤的保护作用。
In the tongue, one of these receptors - called KATP - may help to adjust sensitivity to sweet tastes according to what the body requires.
位于舌头上一种名为KATP的受体能够根据身体的需求程度来调节对甜味的敏感程度。
The sequencing of KATP channel genes, using genomic DNA extracted from the patient's peripheral white blood cells, revealed a genetic mutation.
从病人外周血白细胞中提取出的基因组dna的KATP的基因序列显示其中有一处基因突变。
Excessive activation of KATP is now recognized as a major cause of hypotension and vascular hypo-responsiveness to catecholamine in septic shock.
目前认为。过度激活血管KATP会导致脓毒症体克时的低血压和血管对儿茶酚胺的低反应性。
Conclusion Propofol could decrease blood pressure, which may be due to its effect on the ATP sensitive KATP channels which could inhibit plasma renin activity (PRA) and RAAS.
结论异丙酚降压作用与其作用于KATP通道、抑制肾素活性，进而抑制肾素血管紧张素醛固酮系统有关。
Furthermore, hypothalamic dominant-negative Kir6.2 expression or the delivery of the KATP channel blocker glibenclamide abolished the glucose production-lowering effects of OAG.
此外，下丘脑显性负相Kir2的表达或使用KATP通道阻滞剂格列本脲能抵消oag的作用减少葡萄糖生成的作用。
Furthermore, hypothalamic dominant-negative Kir6.2 expression or the delivery of the KATP channel blocker glibenclamide abolished the glucose production-lowering effects of OAG.
此外，下丘脑显性负相Kir2的表达或使用KATP通道阻滞剂格列本脲能抵消oag的作用减少葡萄糖生成的作用。
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