One of these genes was called “junctional adhesion molecule-1” or JAM-1 for short.
其中一个基因是“接合粘附分子1号（Junctional AdhesionMolecule-1）”，简称为JAM-1。
Uncovering exactly how raised levels of JAM-1 disrupt the brain's ability to control blood pressure is the next step in tackling the disease.
JAM-1分子水平的升高是如何破坏大脑控制血压的能力的呢？
To find out whether this was the cause or effect of high blood pressure, they then transferred the JAM-1 gene into the solitary nucleus of normal rats.
为了查出这是否是高血压的根源，以及高血压的影响，两人把JAM - 1转移到正常老鼠的孤束核中。
They found that in rats with high blood pressure JAM-1 levels were raised not only in the brain but also the kidney, spleen, lungs and skeletal muscle.
Waki和Paton博士发现，老鼠患上高血压后，不仅大脑的JAM - 1水平，而且肾、脾、肺和骨骼肌的JAM - 1水平都升高了。
Variations in the JAM-1 gene and its expression may explain why high blood pressure tends to run in families, although it is also caused by a poor diet.
虽然缺乏饮食也会导致高血压，JAM - 1基因及其表达的差异也可以解释高血压会在家族成员中流传的原因。
Variations in the JAM-1 gene and its expression may explain why high blood pressure tends to run in families, although it is also caused by a poor diet.
虽然缺乏饮食也会导致高血压，JAM - 1基因及其表达的差异也可以解释高血压会在家族成员中流传的原因。
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