Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.
血液学研究显示有严重高磷血症、低钙血症和代谢性酸中毒。
Dietary restriction of phosphate has long been the cornerstone of therapy, but this measure is usually not sufficient to control hyperphosphatemia.
控制磷酸盐摄入成为治疗方法的基石，但这种手段通常不足以控制高磷血症。
Results: the main abnormal blood chemical features in all cases were hyperkalemia, hyperuricemia, hyperphosphatemia and hypocalcemia and abnormal function of the kidney.
结果：所有病例均有不同项目和不同程度的血生化指标异常，主要表现为高钾血症、高尿酸血症、高磷血症和低钙血症以及肾功能异常。
Objective: to investigate the clinical characteristics and mechanism of renal failure due to acute hyperphosphatemia following intravenous fructose diphosphate injection.
目的：探讨注射用二磷酸果糖致急性高磷血症的临床特点及诱发急性肾功能衰竭的发病机理。
Likewise, the presence of hyperphosphatemia, acidosis, and proteinuria may be misleading depending on the experimental model and diet and may not represent a precise measure of progression.
同样地，高磷酸盐血症、酸液过多症、蛋白尿的存在，依靠试验性模型和饮食方式，可能使人误解，并且无法对其扩展性表现出的准确测量。
Likewise, the presence of hyperphosphatemia, acidosis, and proteinuria may be misleading depending on the experimental model and diet and may not represent a precise measure of progression.
同样地，高磷酸盐血症、酸液过多症、蛋白尿的存在，依靠试验性模型和饮食方式，可能使人误解，并且无法对其扩展性表现出的准确测量。
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